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Shao H Yang

Showing results (11-20 of 34) with videos related to

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Transgenic Research|December 19, 2008
Caution! Analyze transcripts from conditional knockout allelesShao H Yang, Martin O Bergo, Emily Farber, et al.
Science (New York, N.Y.)|February 18, 2006
A protein farnesyltransferase inhibitor ameliorates disease in a mouse model of progeriaLoren G Fong, David Frost, Margarita Meta, et al.
Journal of Lipid Research|December 8, 2009
Assessing the efficacy of protein farnesyltransferase inhibitors in mouse models of progeriaShao H Yang, Sandy Y Chang, Douglas A Andres, et al.
Human Molecular Genetics|November 9, 2013
New Lmna knock-in mice provide a molecular mechanism for the 'segmental aging' in Hutchinson-Gilford progeria syndromeHea-Jin Jung, Yiping Tu, Shao H Yang, et al.
Nucleus (Austin, Tex.)|June 8, 2011
Investigating the purpose of prelamin A processingBrandon Sj Davies, Catherine Coffinier, Shao H Yang, et al.
Journal of Lipid Research|November 1, 2011
Severe hepatocellular disease in mice lacking one or both CaaX prenyltransferasesShao H Yang, Sandy Y Chang, Yiping Tu, et al.
Journal of Immunology (Baltimore, Md. : 1950)|January 8, 2008
Requirement of N-myristoyltransferase 1 in the development of monocytic lineageAnuraag Shrivastav, Shailly Varma, Zoe Lawman, et al.
Human Molecular Genetics|November 20, 2010
Absence of progeria-like disease phenotypes in knock-in mice expressing a non-farnesylated version of progerinShao H Yang, Sandy Y Chang, Shuxun Ren, et al.
Molecular and Cellular Biology|October 15, 2014
An absence of nuclear lamins in keratinocytes leads to ichthyosis, defective epidermal barrier function, and intrusion of nuclear membranes and endoplasmic reticulum into the nuclear chromatinHea-Jin Jung, Angelica Tatar, Yiping Tu, et al.
The Journal of Clinical Investigation|July 25, 2006
A farnesyltransferase inhibitor improves disease phenotypes in mice with a Hutchinson-Gilford progeria syndrome mutationShao H Yang, Margarita Meta, Xin Qiao, et al.
Pageof 4

Showing results (11-20 of 34) with videos related to

Sort By:
Pageof 4
Transgenic Research|December 19, 2008
Caution! Analyze transcripts from conditional knockout allelesShao H Yang, Martin O Bergo, Emily Farber, et al.
Science (New York, N.Y.)|February 18, 2006
A protein farnesyltransferase inhibitor ameliorates disease in a mouse model of progeriaLoren G Fong, David Frost, Margarita Meta, et al.
Journal of Lipid Research|December 8, 2009
Assessing the efficacy of protein farnesyltransferase inhibitors in mouse models of progeriaShao H Yang, Sandy Y Chang, Douglas A Andres, et al.
Human Molecular Genetics|November 9, 2013
New Lmna knock-in mice provide a molecular mechanism for the 'segmental aging' in Hutchinson-Gilford progeria syndromeHea-Jin Jung, Yiping Tu, Shao H Yang, et al.
Nucleus (Austin, Tex.)|June 8, 2011
Investigating the purpose of prelamin A processingBrandon Sj Davies, Catherine Coffinier, Shao H Yang, et al.
Journal of Lipid Research|November 1, 2011
Severe hepatocellular disease in mice lacking one or both CaaX prenyltransferasesShao H Yang, Sandy Y Chang, Yiping Tu, et al.
Journal of Immunology (Baltimore, Md. : 1950)|January 8, 2008
Requirement of N-myristoyltransferase 1 in the development of monocytic lineageAnuraag Shrivastav, Shailly Varma, Zoe Lawman, et al.
Human Molecular Genetics|November 20, 2010
Absence of progeria-like disease phenotypes in knock-in mice expressing a non-farnesylated version of progerinShao H Yang, Sandy Y Chang, Shuxun Ren, et al.
Molecular and Cellular Biology|October 15, 2014
An absence of nuclear lamins in keratinocytes leads to ichthyosis, defective epidermal barrier function, and intrusion of nuclear membranes and endoplasmic reticulum into the nuclear chromatinHea-Jin Jung, Angelica Tatar, Yiping Tu, et al.
The Journal of Clinical Investigation|July 25, 2006
A farnesyltransferase inhibitor improves disease phenotypes in mice with a Hutchinson-Gilford progeria syndrome mutationShao H Yang, Margarita Meta, Xin Qiao, et al.
Pageof 4