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Nature Communications|January 9, 2021
Interaction between SNAI2 and MYOD enhances oncogenesis and suppresses differentiation in Fusion Negative RhabdomyosarcomaSilvia Pomella, Prethish Sreenivas, Berkley E Gryder, et al.
Cancer Discovery|April 28, 2017
PAX3-FOXO1 Establishes Myogenic Super Enhancers and Confers BET Bromodomain VulnerabilityBerkley E Gryder, Marielle E Yohe, Hsien-Chao Chou, et al.
Cell Death Discovery|August 6, 2024
HDAC3 genetic and pharmacologic inhibition radiosensitizes fusion positive rhabdomyosarcoma by promoting DNA double-strand breaksMatteo Cassandri, Antonella Porrazzo, Silvia Pomella, et al.
Journal of Hematology & Oncology|December 19, 2024
Tumor-derived G-CSF induces an immunosuppressive microenvironment in an osteosarcoma model, reducing response to CAR.GD2 T-cellsMichele Pezzella, Concetta Quintarelli, Maria C Quadraccia, et al.
Nature Communications|February 24, 2024
KDM3B inhibitors disrupt the oncogenic activity of PAX3-FOXO1 in fusion-positive rhabdomyosarcomaYong Yean Kim, Berkley E Gryder, Ranuka Sinniah, et al.
Nature Communications|December 15, 2023
MYOD-SKP2 axis boosts tumorigenesis in fusion negative rhabdomyosarcoma by preventing differentiation through p57Kip2 targetingSilvia Pomella, Matteo Cassandri, Lucrezia D'Archivio, et al.
Cancers|March 28, 2026
Biological Advances and Current Challenges for Pediatric RhabdomyosarcomaKatie E Hebron, Patience Odeniyide, Yun Wei, et al.
British Journal of Cancer|June 13, 2026
Paediatric Therapeutic Development Workshop on rhabdomyosarcomaJoseph S Baxter, Claudia Montiel Equihua, Jan J Molenaar, et al.
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