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Endocrine-Related Cancer|June 30, 2011
Src kinase activity coordinates cell adhesion and spreading with activation of mammalian target of rapamycin in pancreatic endocrine tumour cellsAlessia Di Florio, Laura Adesso, Simona Pedrotti, et al.Genes & Development|March 20, 2010
MicroRNAs coordinate an alternative splicing network during mouse postnatal heart developmentAuinash Kalsotra, Kun Wang, Pei-Feng Li, et al.Molecular Cell|April 5, 2002
Dynamic antagonism between ETR-3 and PTB regulates cell type-specific alternative splicingNicolas Charlet-B, Penny Logan, Gopal Singh, et al.Human Molecular Genetics|May 16, 2020
Increased nuclear but not cytoplasmic activities of CELF1 protein leads to muscle wastingDiana C Cox, Xiangnan Guan, Zheng Xia, et al.Nucleic Acids Research|February 20, 2004
ETR-3 and CELF4 protein domains required for RNA binding and splicing activity in vivoGopal Singh, Nicolas Charlet-B, Jin Han, et al.Molecular Cell|October 16, 2007
Increased steady-state levels of CUGBP1 in myotonic dystrophy 1 are due to PKC-mediated hyperphosphorylationN Muge Kuyumcu-Martinez, Guey-Shin Wang, Thomas A CooperNucleic Acids Research|November 27, 2010
Identification of MBNL1 and MBNL3 domains required for splicing activation and repressionIoannis Grammatikakis, Young-Hwa Goo, Gloria V Echeverria, et al.Human Molecular Genetics|August 10, 2024
Rescue of Scn5a mis-splicing does not improve the structural and functional heart defects of a DM1 heart mouse modelLarissa Nitschke, Rong-Chi Hu, Andrew N Miller, et al.The Journal of Biological Chemistry|February 6, 2004
CELF6, a member of the CELF family of RNA-binding proteins, regulates muscle-specific splicing enhancer-dependent alternative splicingAndrea N Ladd, Nicole H Nguyen, Kavin Malhotra, et al.Human Molecular Genetics|April 22, 2005
Transgenic mice expressing CUG-BP1 reproduce splicing mis-regulation observed in myotonic dystrophyThai H Ho, Donnie Bundman, Dawna L Armstrong, et al.Pageof 11