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Showing results (141-150 of 220) with videos related to

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The Journal of Endocrinology|February 1, 2017
High fat feeding unmasks variable insulin responses in male C57BL/6 mouse substrainsRebecca L Hull, Joshua R Willard, Matthias D Struck, et al.
Acta Neurochirurgica|August 16, 2002
Decompressive craniectomy for the management of patients with refractory hypertension: should it be reconsidered?V Kontopoulos, N Foroglou, J Patsalas, et al.
Diabetologia|May 2, 2020
E2f8 and Dlg2 genes have independent effects on impaired insulin secretion associated with hyperglycaemiaChieh-Hsin Yang, Salvatore P Mangiafico, Michaela Waibel, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|February 24, 2007
Metabolism and anticancer activity of the curcumin analogue, dimethoxycurcuminConstantin Tamvakopoulos, Konstantinos Dimas, Zacharias D Sofianos, et al.
Endocrinology|May 22, 2002
Comparison of insulin secretory function in two mouse models with different susceptibility to beta-cell failureSuwattanee Kooptiwut, Sakeneh Zraika, Anne W Thorburn, et al.
Endocrinology|July 28, 2011
Deficiency in interferon-gamma results in reduced body weight and better glucose tolerance in miceNicole Wong, Barbara C Fam, Gitta R Cempako, et al.
Molecular Endocrinology (Baltimore, Md.)|January 28, 2012
The nuclear receptor, Nor-1, markedly increases type II oxidative muscle fibers and resistance to fatigueMichael A Pearen, Natalie A Eriksson, Rebecca L Fitzsimmons, et al.
Diabetes|January 24, 2003
Increased dietary fat promotes islet amyloid formation and beta-cell secretory dysfunction in a transgenic mouse model of islet amyloidRebecca L Hull, Sofianos Andrikopoulos, C Bruce Verchere, et al.
Physiological Reports|April 19, 2022
Blood glucose modulation and safety of efferent vagus nerve stimulation in a type 2 diabetic rat modelSophie C Payne, Glenn Ward, James B Fallon, et al.
The Journal of Endocrinology|June 25, 2011
A primary defect in glucose production alone cannot induce glucose intolerance without defects in insulin secretionSalvatore P Mangiafico, Shueh H Lim, Sandra Neoh, et al.
Pageof 22

Showing results (141-150 of 220) with videos related to

Sort By:
Pageof 22
The Journal of Endocrinology|February 1, 2017
High fat feeding unmasks variable insulin responses in male C57BL/6 mouse substrainsRebecca L Hull, Joshua R Willard, Matthias D Struck, et al.
Acta Neurochirurgica|August 16, 2002
Decompressive craniectomy for the management of patients with refractory hypertension: should it be reconsidered?V Kontopoulos, N Foroglou, J Patsalas, et al.
Diabetologia|May 2, 2020
E2f8 and Dlg2 genes have independent effects on impaired insulin secretion associated with hyperglycaemiaChieh-Hsin Yang, Salvatore P Mangiafico, Michaela Waibel, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|February 24, 2007
Metabolism and anticancer activity of the curcumin analogue, dimethoxycurcuminConstantin Tamvakopoulos, Konstantinos Dimas, Zacharias D Sofianos, et al.
Endocrinology|May 22, 2002
Comparison of insulin secretory function in two mouse models with different susceptibility to beta-cell failureSuwattanee Kooptiwut, Sakeneh Zraika, Anne W Thorburn, et al.
Endocrinology|July 28, 2011
Deficiency in interferon-gamma results in reduced body weight and better glucose tolerance in miceNicole Wong, Barbara C Fam, Gitta R Cempako, et al.
Molecular Endocrinology (Baltimore, Md.)|January 28, 2012
The nuclear receptor, Nor-1, markedly increases type II oxidative muscle fibers and resistance to fatigueMichael A Pearen, Natalie A Eriksson, Rebecca L Fitzsimmons, et al.
Diabetes|January 24, 2003
Increased dietary fat promotes islet amyloid formation and beta-cell secretory dysfunction in a transgenic mouse model of islet amyloidRebecca L Hull, Sofianos Andrikopoulos, C Bruce Verchere, et al.
Physiological Reports|April 19, 2022
Blood glucose modulation and safety of efferent vagus nerve stimulation in a type 2 diabetic rat modelSophie C Payne, Glenn Ward, James B Fallon, et al.
The Journal of Endocrinology|June 25, 2011
A primary defect in glucose production alone cannot induce glucose intolerance without defects in insulin secretionSalvatore P Mangiafico, Shueh H Lim, Sandra Neoh, et al.
Pageof 22