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Steven J Coultrap

Showing results (11-20 of 45) with videos related to

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Science Advances|April 15, 2021
CaMKII holoenzyme mechanisms that govern the LTP versus LTD decisionSarah G Cook, Olivia R Buonarati, Steven J Coultrap, et al.
Brain Research|June 15, 2007
alphaCaMKII autophosphorylation levels differ depending on subcellular localizationKurtis D Davies, Rachel M Alvestad, Steven J Coultrap, et al.
Plos One|May 7, 2014
CaMKII binding to GluN2B is differentially affected by macromolecular crowding reagentsDayton J Goodell, Tatiana A Eliseeva, Steven J Coultrap, et al.
The Journal of Biological Chemistry|September 30, 2008
Long term synaptic depression that is associated with GluR1 dephosphorylation but not alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor internalizationKurtis D Davies, Susan M Goebel-Goody, Steven J Coultrap, et al.
Acta Pharmacologica Sinica|June 21, 2011
CaMKII in cerebral ischemiaSteven J Coultrap, Rebekah S Vest, Nicole M Ashpole, et al.
The Journal of Biological Chemistry|July 20, 2011
Nucleotides and phosphorylation bi-directionally modulate Ca2+/calmodulin-dependent protein kinase II (CaMKII) binding to the N-methyl-D-aspartate (NMDA) receptor subunit GluN2BHeather O'Leary, Wallace H Liu, Jacki M Rorabaugh, et al.
The Journal of Biological Chemistry|April 1, 2010
CaMKII autonomy is substrate-dependent and further stimulated by Ca2+/calmodulinSteven J Coultrap, Isabelle Buard, Jaqueline R Kulbe, et al.
Cell Reports|June 15, 2017
DAPK1 Mediates LTD by Making CaMKII/GluN2B Binding LTP SpecificDayton J Goodell, Vincent Zaegel, Steven J Coultrap, et al.
The Journal of Biological Chemistry|April 29, 2010
Effective post-insult neuroprotection by a novel Ca(2+)/ calmodulin-dependent protein kinase II (CaMKII) inhibitorRebekah S Vest, Heather O'Leary, Steven J Coultrap, et al.
Molecular Pharmacology|April 4, 2025
The neuroprotective γ-hydroxybutyrate analog 3-hydroxycyclopent-1-enecarboxylic acid does not directly affect CaMKIIα autophosphorylation at T286 or binding to GluN2BCarolyn Nicole Brown, Rachel E Blaine, Chase Madison Barker, et al.
Pageof 5

Showing results (11-20 of 45) with videos related to

Sort By:
Pageof 5
Science Advances|April 15, 2021
CaMKII holoenzyme mechanisms that govern the LTP versus LTD decisionSarah G Cook, Olivia R Buonarati, Steven J Coultrap, et al.
Brain Research|June 15, 2007
alphaCaMKII autophosphorylation levels differ depending on subcellular localizationKurtis D Davies, Rachel M Alvestad, Steven J Coultrap, et al.
Plos One|May 7, 2014
CaMKII binding to GluN2B is differentially affected by macromolecular crowding reagentsDayton J Goodell, Tatiana A Eliseeva, Steven J Coultrap, et al.
The Journal of Biological Chemistry|September 30, 2008
Long term synaptic depression that is associated with GluR1 dephosphorylation but not alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor internalizationKurtis D Davies, Susan M Goebel-Goody, Steven J Coultrap, et al.
Acta Pharmacologica Sinica|June 21, 2011
CaMKII in cerebral ischemiaSteven J Coultrap, Rebekah S Vest, Nicole M Ashpole, et al.
The Journal of Biological Chemistry|July 20, 2011
Nucleotides and phosphorylation bi-directionally modulate Ca2+/calmodulin-dependent protein kinase II (CaMKII) binding to the N-methyl-D-aspartate (NMDA) receptor subunit GluN2BHeather O'Leary, Wallace H Liu, Jacki M Rorabaugh, et al.
The Journal of Biological Chemistry|April 1, 2010
CaMKII autonomy is substrate-dependent and further stimulated by Ca2+/calmodulinSteven J Coultrap, Isabelle Buard, Jaqueline R Kulbe, et al.
Cell Reports|June 15, 2017
DAPK1 Mediates LTD by Making CaMKII/GluN2B Binding LTP SpecificDayton J Goodell, Vincent Zaegel, Steven J Coultrap, et al.
The Journal of Biological Chemistry|April 29, 2010
Effective post-insult neuroprotection by a novel Ca(2+)/ calmodulin-dependent protein kinase II (CaMKII) inhibitorRebekah S Vest, Heather O'Leary, Steven J Coultrap, et al.
Molecular Pharmacology|April 4, 2025
The neuroprotective γ-hydroxybutyrate analog 3-hydroxycyclopent-1-enecarboxylic acid does not directly affect CaMKIIα autophosphorylation at T286 or binding to GluN2BCarolyn Nicole Brown, Rachel E Blaine, Chase Madison Barker, et al.
Pageof 5