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Channels (Austin, Tex.)|August 12, 2010
Splice-variant changes of the Ca(V)3.2 T-type calcium channel mediate voltage-dependent facilitation and associate with cardiac hypertrophy and developmentLaurence S David, Esperanza Garcia, Stuart M Cain, et al.Neurorx : the Journal of the American Society for Experimental Neurotherapeutics|February 21, 2006
Targeting chronic and neuropathic pain: the N-type calcium channel comes of ageTerrance P SnutchThe European Journal of Neuroscience|October 23, 2015
The Genetic Absence Epilepsy Rats from Strasbourg model of absence epilepsy exhibits alterations in fear conditioning and latent inhibition consistent with psychiatric comorbidities in humansWendie N Marks, Mary E Cavanagh, Quentin Greba, et al.The European Journal of Neuroscience|March 20, 2019
The T-type calcium channel blocker Z944 reduces conditioned fear in Genetic Absence Epilepsy Rats from Strasbourg and the non-epileptic control strainWendie N Marks, Nadine K Zabder, Quentin Greba, et al.Cell Calcium|September 28, 2014
Epigallocatechin-3-gallate elicits Ca2+ spike in MCF-7 breast cancer cells: essential role of Cav3.2 channelsElia Ranzato, Valeria Magnelli, Simona Martinotti, et al.Experimental Neurology|June 29, 2017
Sociability impairments in Genetic Absence Epilepsy Rats from Strasbourg: Reversal by the T-type calcium channel antagonist Z944Mark T Henbid, Wendie N Marks, Madeline J Collins, et al.Scientific Reports|September 16, 2017
The Cacna1h mutation in the GAERS model of absence epilepsy enhances T-type Ca2+ currents by altering calnexin-dependent trafficking of Cav3.2 channelsJuliane Proft, Yuriy Rzhepetskyy, Joanna Lazniewska, et al.Molecular Interventions|March 3, 2004
Modulating modulation: crosstalk between regulatory pathways of presynaptic calcium channelsGerald W Zamponi, Terrance P SnutchPageof 13