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Sylke Lutz

Showing results (1-10 of 5) with videos related to

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BMC Developmental Biology|April 29, 2008
Expression patterns of protein kinase D 3 during mouse developmentKornelia Ellwanger, Klaus Pfizenmaier, Sylke Lutz, et al.
The Journal of Cell Biology|January 5, 2002
Structural requirements for localization and activation of protein kinase C mu (PKC mu) at the Golgi compartmentAngelika Hausser, Gisela Link, Linda Bamberg, et al.
The Biochemical Journal|August 19, 2011
Protein kinase D controls voluntary-running-induced skeletal muscle remodellingKornelia Ellwanger, Christine Kienzle, Sylke Lutz, et al.
Molecular Biology of the Cell|February 13, 2009
Protein kinase D controls the integrity of Golgi apparatus and the maintenance of dendritic arborization in hippocampal neuronsKatalin Czöndör, Kornelia Ellwanger, Yannick F Fuchs, et al.
Molecular Metabolism|December 5, 2025
Protein kinase D deficiency induces a senescence-like phenotype in β-cells and improves glucose and insulin tolerance under high-fat diet conditionsWolfgang S Lieb, Carlos O Oueslati Morales, Kornelia Ellwanger, et al.
Pageof 1

Showing results (1-10 of 5) with videos related to

Sort By:
Pageof 1
BMC Developmental Biology|April 29, 2008
Expression patterns of protein kinase D 3 during mouse developmentKornelia Ellwanger, Klaus Pfizenmaier, Sylke Lutz, et al.
The Journal of Cell Biology|January 5, 2002
Structural requirements for localization and activation of protein kinase C mu (PKC mu) at the Golgi compartmentAngelika Hausser, Gisela Link, Linda Bamberg, et al.
The Biochemical Journal|August 19, 2011
Protein kinase D controls voluntary-running-induced skeletal muscle remodellingKornelia Ellwanger, Christine Kienzle, Sylke Lutz, et al.
Molecular Biology of the Cell|February 13, 2009
Protein kinase D controls the integrity of Golgi apparatus and the maintenance of dendritic arborization in hippocampal neuronsKatalin Czöndör, Kornelia Ellwanger, Yannick F Fuchs, et al.
Molecular Metabolism|December 5, 2025
Protein kinase D deficiency induces a senescence-like phenotype in β-cells and improves glucose and insulin tolerance under high-fat diet conditionsWolfgang S Lieb, Carlos O Oueslati Morales, Kornelia Ellwanger, et al.
Pageof 1