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Cell Calcium|October 5, 2025
Pathological calcium influx through amyloid beta pores disrupts synaptic functionTemitope Adeoye, Ghanim UllahBiorxiv : the Preprint Server for Biology|April 1, 2024
Systematic Analysis of Biological Processes Reveals Gene Co-expression Modules Driving Pathway Dysregulation in Alzheimer's DiseaseTemitope Adeoye, Syed I Shah, Ghanim UllahAging and Disease|June 24, 2024
Systematic Analysis of Biological Processes Reveals Gene Co-expression Modules Driving Pathway Dysregulation in Alzheimer's DiseaseTemitope Adeoye, Syed I Shah, Ghanim UllahCells|July 27, 2022
Upregulated Ca<sup>2+</sup> Release from the Endoplasmic Reticulum Leads to Impaired Presynaptic Function in Familial Alzheimer's DiseaseTemitope Adeoye, Syed I Shah, Angelo Demuro, et al.Journal of Computational Neuroscience|September 12, 2019
Mitochondrial dysfunction and role in spreading depolarization and seizurePatrick Toglia, Ghanim UllahEuropean Biophysics Journal : EBJ|February 11, 2018
Reduced cooperativity of voltage-gated sodium channels in the hippocampal interneurons of an aged mouse model of Alzheimer's diseaseCarlos Perez, Ghanim UllahPlos One|March 15, 2016
Anions Govern Cell Volume: A Case Study of Relative Astrocytic and Neuronal Swelling in Spreading DepolarizationNiklas Hübel, Ghanim UllahCell Calcium|May 18, 2016
The gain-of-function enhancement of IP3-receptor channel gating by familial Alzheimer's disease-linked presenilin mutants increases the open probability of mitochondrial permeability transition porePatrick Toglia, Ghanim UllahJournal of Biological Physics|May 8, 2016
Mode switching of Inositol 1,4,5-trisphosphate receptor channel shapes the Spatiotemporal scales of Ca<sup>2+</sup> signalsGhanim Ullah, Aman UllahBiophysical Journal|March 4, 2006
Modeling the statistics of elementary calcium release eventsGhanim Ullah, Peter JungPageof 7