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Thomas M Roberts

Showing results (41-50 of 127) with videos related to

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Iscience|November 5, 2018
A Conditional Dependency on MELK for the Proliferation of Triple-Negative Breast Cancer CellsYubao Wang, Ben B Li, Jing Li, et al.
Journal of Molecular Biology|March 10, 2005
Structure of MFP2 and its function in enhancing MSP polymerization in Ascaris sperm amoeboid motilityRichard P Grant, Shawnna M Buttery, Gail C Ekman, et al.
The Journal of Biological Chemistry|January 5, 2002
DEF-1/ASAP1 is a GTPase-activating protein (GAP) for ARF1 that enhances cell motility through a GAP-dependent mechanismCraig Furman, Sarah M Short, Romesh R Subramanian, et al.
Journal of Virology|August 13, 2011
Comparisons between murine polyomavirus and Simian virus 40 show significant differences in small T antigen functionShaida Andrabi, Justin H Hwang, Jennifer Kean Choe, et al.
Molecular Biology of the Cell|May 22, 2009
Dephosphorylation of major sperm protein (MSP) fiber protein 3 by protein phosphatase 2A during cell body retraction in the MSP-based amoeboid motility of Ascaris spermKexi Yi, Xu Wang, Mark R Emmett, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 13, 2005
The oncogenic properties of mutant p110alpha and p110beta phosphatidylinositol 3-kinases in human mammary epithelial cellsJean J Zhao, Zhenning Liu, Li Wang, et al.
Journal of Virology|July 4, 2014
The phosphatidylinositol 3-kinase (PI3K) isoform dependence of tumor formation is determined by the genetic mode of PI3K pathway activation rather than by tissue typeTamara Utermark, Fabienne Schmit, Sang Hyun Lee, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 15, 2020
PIK3CA C-terminal frameshift mutations are novel oncogenic events that sensitize tumors to PI3K-α inhibitionJennifer M Spangle, Thanh Von, Dean C Pavlick, et al.
Scientific Reports|January 12, 2021
The role of the PIK3CA gene in the development and aging of the brainShaozhen Xie, Jing Ni, Hanbing Guo, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 17, 2014
PI3K isoform dependence of PTEN-deficient tumors can be altered by the genetic contextFabienne Schmit, Tamara Utermark, Sen Zhang, et al.
Pageof 13

Showing results (41-50 of 127) with videos related to

Sort By:
Pageof 13
Iscience|November 5, 2018
A Conditional Dependency on MELK for the Proliferation of Triple-Negative Breast Cancer CellsYubao Wang, Ben B Li, Jing Li, et al.
Journal of Molecular Biology|March 10, 2005
Structure of MFP2 and its function in enhancing MSP polymerization in Ascaris sperm amoeboid motilityRichard P Grant, Shawnna M Buttery, Gail C Ekman, et al.
The Journal of Biological Chemistry|January 5, 2002
DEF-1/ASAP1 is a GTPase-activating protein (GAP) for ARF1 that enhances cell motility through a GAP-dependent mechanismCraig Furman, Sarah M Short, Romesh R Subramanian, et al.
Journal of Virology|August 13, 2011
Comparisons between murine polyomavirus and Simian virus 40 show significant differences in small T antigen functionShaida Andrabi, Justin H Hwang, Jennifer Kean Choe, et al.
Molecular Biology of the Cell|May 22, 2009
Dephosphorylation of major sperm protein (MSP) fiber protein 3 by protein phosphatase 2A during cell body retraction in the MSP-based amoeboid motility of Ascaris spermKexi Yi, Xu Wang, Mark R Emmett, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 13, 2005
The oncogenic properties of mutant p110alpha and p110beta phosphatidylinositol 3-kinases in human mammary epithelial cellsJean J Zhao, Zhenning Liu, Li Wang, et al.
Journal of Virology|July 4, 2014
The phosphatidylinositol 3-kinase (PI3K) isoform dependence of tumor formation is determined by the genetic mode of PI3K pathway activation rather than by tissue typeTamara Utermark, Fabienne Schmit, Sang Hyun Lee, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 15, 2020
PIK3CA C-terminal frameshift mutations are novel oncogenic events that sensitize tumors to PI3K-α inhibitionJennifer M Spangle, Thanh Von, Dean C Pavlick, et al.
Scientific Reports|January 12, 2021
The role of the PIK3CA gene in the development and aging of the brainShaozhen Xie, Jing Ni, Hanbing Guo, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 17, 2014
PI3K isoform dependence of PTEN-deficient tumors can be altered by the genetic contextFabienne Schmit, Tamara Utermark, Sen Zhang, et al.
Pageof 13