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Tim Thomas

Showing results (91-100 of 147) with videos related to

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Development (Cambridge, England)|July 26, 2019
MOZ directs the distal-less homeobox gene expression program during craniofacial developmentHannah K Vanyai, Alexandra Garnham, Rose E May, et al.
Development (Cambridge, England)|June 2, 2026
The roles of the acetyltransferase domains of the KAT6A and KAT6B in vivoTim Thomas, Shezlie Malelang, Yuqing Yang, et al.
Molecular and Cellular Biology|November 27, 2019
HBO1 (KAT7) Does Not Have an Essential Role in Cell Proliferation, DNA Replication, or Histone 4 Acetylation in Human CellsAndrew J Kueh, Samantha Eccles, Leonie Tang, et al.
Blood|September 25, 2016
MOZ (KAT6A) is essential for the maintenance of classically defined adult hematopoietic stem cellsBilal N Sheikh, Yuqing Yang, Jaring Schreuder, et al.
Developmental Biology|January 20, 2025
Loss of KAT6B causes premature ossification and promotes osteoblast differentiation during developmentMaria I Bergamasco, Jacqueline M Ogier, Alexandra L Garnham, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 30, 2015
MOZ and BMI1 play opposing roles during Hox gene activation in ES cells and in body segment identity specification in vivoBilal N Sheikh, Natalie L Downer, Belinda Phipson, et al.
Life Science Alliance|November 13, 2024
KAT6B is required for histone 3 lysine 9 acetylation and SOX gene expression in the developing brainMaria I Bergamasco, Waruni Abeysekera, Alexandra L Garnham, et al.
Plos Genetics|May 4, 2026
KAT6A is essential for developmental control gene expression in neural stem and progenitor cellsAnne K Voss, Samantha Eccles, Johannes Wichmann, et al.
Journal of Cell Science|May 31, 2007
Hrk/DP5 contributes to the apoptosis of select neuronal populations but is dispensable for haematopoietic cell apoptosisLeigh Coultas, Susanna Terzano, Tim Thomas, et al.
Genomics Data|October 21, 2015
Transcriptional profiling of the postnatal brain of the Ts1Cje mouse model of Down syndromeKai-Leng Tan, King-Hwa Ling, Chelsee A Hewitt, et al.
Pageof 15

Showing results (91-100 of 147) with videos related to

Sort By:
Pageof 15
Development (Cambridge, England)|July 26, 2019
MOZ directs the distal-less homeobox gene expression program during craniofacial developmentHannah K Vanyai, Alexandra Garnham, Rose E May, et al.
Development (Cambridge, England)|June 2, 2026
The roles of the acetyltransferase domains of the KAT6A and KAT6B in vivoTim Thomas, Shezlie Malelang, Yuqing Yang, et al.
Molecular and Cellular Biology|November 27, 2019
HBO1 (KAT7) Does Not Have an Essential Role in Cell Proliferation, DNA Replication, or Histone 4 Acetylation in Human CellsAndrew J Kueh, Samantha Eccles, Leonie Tang, et al.
Blood|September 25, 2016
MOZ (KAT6A) is essential for the maintenance of classically defined adult hematopoietic stem cellsBilal N Sheikh, Yuqing Yang, Jaring Schreuder, et al.
Developmental Biology|January 20, 2025
Loss of KAT6B causes premature ossification and promotes osteoblast differentiation during developmentMaria I Bergamasco, Jacqueline M Ogier, Alexandra L Garnham, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 30, 2015
MOZ and BMI1 play opposing roles during Hox gene activation in ES cells and in body segment identity specification in vivoBilal N Sheikh, Natalie L Downer, Belinda Phipson, et al.
Life Science Alliance|November 13, 2024
KAT6B is required for histone 3 lysine 9 acetylation and SOX gene expression in the developing brainMaria I Bergamasco, Waruni Abeysekera, Alexandra L Garnham, et al.
Plos Genetics|May 4, 2026
KAT6A is essential for developmental control gene expression in neural stem and progenitor cellsAnne K Voss, Samantha Eccles, Johannes Wichmann, et al.
Journal of Cell Science|May 31, 2007
Hrk/DP5 contributes to the apoptosis of select neuronal populations but is dispensable for haematopoietic cell apoptosisLeigh Coultas, Susanna Terzano, Tim Thomas, et al.
Genomics Data|October 21, 2015
Transcriptional profiling of the postnatal brain of the Ts1Cje mouse model of Down syndromeKai-Leng Tan, King-Hwa Ling, Chelsee A Hewitt, et al.
Pageof 15