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Bio-Medical Materials and Engineering|July 20, 2024
Reliability and validity of estimated angles information assessed using inertial measurement unit-based motion sensorsTaiki Morikawa, Nariyuki Mura, Toshiaki Sato, et al.Diabetes/Metabolism Research and Reviews|January 31, 2006
Effects of sulfonylureas on mitochondrial ATP-sensitive K+ channels in cardiac myocytes: implications for sulfonylurea controversyToshiaki Sato, Hirofumi Nishida, Masaru Miyazaki, et al.Journal of Pharmacological Sciences|March 10, 2009
New aspects for the treatment of cardiac diseases based on the diversity of functional controls on cardiac muscles: mitochondrial ion channels and cardioprotectionHirofumi Nishida, Toshiaki Sato, Takehiko Ogura, et al.BMC Sports Science, Medicine & Rehabilitation|July 17, 2024
Validity of the estimated angular information obtained using an inertial motion capture system during standing trunk forward and backward bendingTaiki Morikawa, Nariyuki Mura, Toshiaki Sato, et al.Journal of Arrhythmia|June 29, 2018
Subclinical cardiac perforation caused by a Micra™ leadless pacemakerIkuko Togashi, Toshiaki Sato, Kyoko Hoshida, et al.Circulation|December 30, 2004
Mitochondrial Ca2+-activated K+ channels in cardiac myocytes: a mechanism of the cardioprotective effect and modulation by protein kinase AToshiaki Sato, Tomoaki Saito, Noriko Saegusa, et al.American Journal of Physiology. Heart and Circulatory Physiology|March 8, 2003
Diazoxide triggers cardioprotection against apoptosis induced by oxidative stressMasashi Ichinose, Hidetoshi Yonemochi, Toshiaki Sato, et al.Naunyn-Schmiedeberg'S Archives of Pharmacology|November 24, 2004
Inhibitory effects of AMP 579, a novel cardioprotective adenosine A1/A2A receptor agonist, on native IKr and cloned HERG currentNoriko Saegusa, Toshiaki Sato, Takehiko Ogura, et al.Drug, Healthcare and Patient Safety|June 25, 2011
A danger of induction of Brugada syndrome during pill-in-the-pocket therapy for paroxysmal atrial fibrillationYoshiyasu Aizawa, Tomohiro Matsuhashi, Toshiaki Sato, et al.The Journal of Pharmacology and Experimental Therapeutics|April 3, 2008
6-[4-(1-Cyclohexyl-1H-tetrazol-5-yl)butoxy]-3,4-dihydro-2-(1H)quinolinone (cilostazol), a phosphodiesterase type 3 inhibitor, reduces infarct size via activation of mitochondrial Ca2+-activated K+ channels in rabbit heartsMika Fukasawa, Hirofumi Nishida, Toshiaki Sato, et al.Pageof 12