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Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|December 8, 2016
Predicting MEK Inhibitor Response in Lung Cancer: A Proper Signature Is RequiredYaohua Xue, Piro LitoCancer Cell|March 12, 2024
Insights into how adeno-squamous transition drives KRAS inhibitor resistanceFeng Hu, Piro LitoNature|January 10, 2020
Rapid non-uniform adaptation to conformation-specific KRAS(G12C) inhibitionJenny Y Xue, Yulei Zhao, Jordan Aronowitz, et al.Cancer Discovery|May 18, 2021
Suppressing Nucleotide Exchange to Inhibit KRAS-Mutant TumorsYulei Zhao, Jenny Y Xue, Piro LitoNature Medicine|November 9, 2013
Tumor adaptation and resistance to RAF inhibitorsPiro Lito, Neal Rosen, David B SolitCell|October 16, 2020
Targeting KRAS(G12C): From Inhibitory Mechanism to Modulation of Antitumor Effects in PatientsDongsung Kim, Jenny Yaohua Xue, Piro LitoScience (New York, N.Y.)|February 4, 2016
Allele-specific inhibitors inactivate mutant KRAS G12C by a trapping mechanismPiro Lito, Martha Solomon, Lian-Sheng Li, et al.Science (New York, N.Y.)|October 7, 2021
The G protein signaling regulator RGS3 enhances the GTPase activity of KRASChuanchuan Li, Alberto Vides, Dongsung Kim, et al.The Journal of Biological Chemistry|November 15, 2008
Sprouty 2 regulates DNA damage-induced apoptosis in Ras-transformed human fibroblastsPiro Lito, Bryan D Mets, Daniel M Appledorn, et al.The Journal of Biological Chemistry|December 1, 2007
Evidence that sprouty 2 is necessary for sarcoma formation by H-Ras oncogene-transformed human fibroblastsPiro Lito, Bryan D Mets, Susanne Kleff, et al.Pageof 5