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Trevi A Ramirez

Showing results (1-10 of 8) with videos related to

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Hypertension Research : Official Journal of the Japanese Society of Hypertension|April 13, 2012
Chronic and intermittent hypoxia differentially regulate left ventricular inflammatory and extracellular matrix responsesTrevi A Ramirez, Claude Jourdan-Le Saux, Anne Joy, et al.
Cardiovascular Research|August 25, 2012
Matrix metalloproteinase-9 deletion attenuates myocardial fibrosis and diastolic dysfunction in ageing miceYing Ann Chiao, Trevi A Ramirez, Rogelio Zamilpa, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 24, 2011
SPARC mediates early extracellular matrix remodeling following myocardial infarctionSarah M McCurdy, Qiuxia Dai, Jianhua Zhang, et al.
Circulation Research|December 25, 2012
Matrix metalloproteinase-28 deletion exacerbates cardiac dysfunction and rupture after myocardial infarction in mice by inhibiting M2 macrophage activationYonggang Ma, Ganesh V Halade, Jianhua Zhang, et al.
American Journal of Physiology. Heart and Circulatory Physiology|October 22, 2013
Reduced BDNF attenuates inflammation and angiogenesis to improve survival and cardiac function following myocardial infarction in miceGanesh V Halade, Yonggang Ma, Trevi A Ramirez, et al.
Journal of Molecular and Cellular Cardiology|April 29, 2014
Aliskiren and valsartan mediate left ventricular remodeling post-myocardial infarction in mice through MMP-9 effectsTrevi A Ramirez, Rugmani Padmanabhan Iyer, Omid Ghasemi, et al.
Journal of Proteomics|May 18, 2013
Texas 3-step decellularization protocol: looking at the cardiac extracellular matrixLisandra E de Castro Brás, Trevi A Ramirez, Kristine Y DeLeon-Pennell, et al.
Journal of Molecular and Cellular Cardiology|August 14, 2012
Transgenic overexpression of matrix metalloproteinase-9 in macrophages attenuates the inflammatory response and improves left ventricular function post-myocardial infarctionRogelio Zamilpa, Jessica Ibarra, Lisandra E de Castro Brás, et al.
Pageof 1

Showing results (1-10 of 8) with videos related to

Sort By:
Pageof 1
Hypertension Research : Official Journal of the Japanese Society of Hypertension|April 13, 2012
Chronic and intermittent hypoxia differentially regulate left ventricular inflammatory and extracellular matrix responsesTrevi A Ramirez, Claude Jourdan-Le Saux, Anne Joy, et al.
Cardiovascular Research|August 25, 2012
Matrix metalloproteinase-9 deletion attenuates myocardial fibrosis and diastolic dysfunction in ageing miceYing Ann Chiao, Trevi A Ramirez, Rogelio Zamilpa, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 24, 2011
SPARC mediates early extracellular matrix remodeling following myocardial infarctionSarah M McCurdy, Qiuxia Dai, Jianhua Zhang, et al.
Circulation Research|December 25, 2012
Matrix metalloproteinase-28 deletion exacerbates cardiac dysfunction and rupture after myocardial infarction in mice by inhibiting M2 macrophage activationYonggang Ma, Ganesh V Halade, Jianhua Zhang, et al.
American Journal of Physiology. Heart and Circulatory Physiology|October 22, 2013
Reduced BDNF attenuates inflammation and angiogenesis to improve survival and cardiac function following myocardial infarction in miceGanesh V Halade, Yonggang Ma, Trevi A Ramirez, et al.
Journal of Molecular and Cellular Cardiology|April 29, 2014
Aliskiren and valsartan mediate left ventricular remodeling post-myocardial infarction in mice through MMP-9 effectsTrevi A Ramirez, Rugmani Padmanabhan Iyer, Omid Ghasemi, et al.
Journal of Proteomics|May 18, 2013
Texas 3-step decellularization protocol: looking at the cardiac extracellular matrixLisandra E de Castro Brás, Trevi A Ramirez, Kristine Y DeLeon-Pennell, et al.
Journal of Molecular and Cellular Cardiology|August 14, 2012
Transgenic overexpression of matrix metalloproteinase-9 in macrophages attenuates the inflammatory response and improves left ventricular function post-myocardial infarctionRogelio Zamilpa, Jessica Ibarra, Lisandra E de Castro Brás, et al.
Pageof 1