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Proceedings of the Association of American Physicians|February 14, 1998
Homocysteine-induced nitric oxide production in vascular smooth-muscle cells by NF-kappa B-dependent transcriptional activation of Nos2G N Welch, G R Upchurch, R S Farivar, et al.Current Opinion in Cardiology|February 24, 2001
Roles of endothelial dysfunction in coronary artery diseaseM A Forgione, J A Leopold, J LoscalzoCirculation Research|November 11, 2006
Vascular calcification: pathobiological mechanisms and clinical implicationsRebecca C Johnson, Jane A Leopold, Joseph LoscalzoCongestive Heart Failure (Greenwich, Conn.)|April 30, 2005
Genetic determinants of vascular oxidant stress and endothelial dysfunctionJoseph Loscalzo, Barbara Voetsch, Ronglih Liao, et al.IUBMB Life|November 23, 2013
Systems analysis of oxidant stress in the vasculatureDiane E Handy, Joseph Loscalzo, Jane A LeopoldProgress in Cardiovascular Diseases|May 25, 2002
Atherothrombosis: plaque instability and thrombogenesisFrederick L Ruberg, Jane A Leopold, Joseph LoscalzoThe Journal of Clinical Investigation|January 3, 2020
The application of big data to cardiovascular disease: paths to precision medicineJane A Leopold, Bradley A Maron, Joseph LoscalzoFASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|July 9, 2020
Network determinants of cardiovascular calcification and repositioned drug treatmentsEuijun Song, Rui-Sheng Wang, Jane A Leopold, et al.Journal of Cardiovascular Pharmacology and Therapeutics|August 18, 2001
Heparin reacts with and inactivates nitric oxideG R Upchurch, D G Goodman, S R Willoughby, et al.Circulation|April 15, 1997
High-dose heparin decreases nitric oxide production by cultured bovine endothelial cellsG R Upchurch, G N Welch, J E Freedman, et al.Pageof 1,828