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Rheumatology (Oxford, England)|April 3, 2014
Mucocutaneous manifestations in a UK national cohort of juvenile-onset systemic lupus erythematosus patientsDirekrit Chiewchengchol, Ruth Murphy, Thomas Morgan, et al.
Rheumatology (Oxford, England)|March 10, 2021
Limited sensitivity and specificity of the ACR/EULAR-2019 classification criteria for SLE in JSLE?-observations from the UK JSLE Cohort StudyEve M D Smith, Sajida Rasul, Coziana Ciurtin, et al.
Rheumatology (Oxford, England)|May 9, 2022
Panel sequencing links rare, likely damaging gene variants with distinct clinical phenotypes and outcomes in juvenile-onset SLEAmandine Charras, Sam Haldenby, Eve M D Smith, et al.
Clinical Immunology (Orlando, Fla.)|May 5, 2022
Real world treatment of juvenile-onset systemic lupus erythematosus: Data from the UK JSLE cohort studyEve M D Smith, Naomi Egbivwie, Andrea L Jorgensen, et al.
Pediatric Rheumatology Online Journal|May 1, 2025
Developing consensus outcome measures in juvenile systemic sclerosis: a global survey of pediatric rheumatologists and literature reviewNatalia Vasquez Canizares, Clare E Pain, Francesco Zulian, et al.
Nature Communications|February 19, 2025
A decision point between transdifferentiation and programmed cell death priming controls KRAS-dependent pancreatic cancer developmentAnne T Schneider, Christiane Koppe, Emilie Crouchet, et al.
Cancer Cell|June 14, 2017
Kupffer Cell-Derived Tnf Triggers Cholangiocellular Tumorigenesis through JNK due to Chronic Mitochondrial Dysfunction and ROSDetian Yuan, Shan Huang, Emanuel Berger, et al.
Nature|March 25, 2021
Auto-aggressive CXCR6<sup>+</sup> CD8 T cells cause liver immune pathology in NASHMichael Dudek, Dominik Pfister, Sainitin Donakonda, et al.
Nature Medicine|April 3, 2019
Platelet GPIbα is a mediator and potential interventional target for NASH and subsequent liver cancerMohsen Malehmir, Dominik Pfister, Suchira Gallage, et al.
Journal of Hepatology|May 24, 2023
Intestinal B cells license metabolic T-cell activation in NASH microbiota/antigen-independently and contribute to fibrosis by IgA-FcR signallingElena Kotsiliti, Valentina Leone, Svenja Schuehle, et al.
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