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Journal of Visualized Experiments : Jove|June 30, 2020
Measuring the Confluence of iPSCs using an Automated Imaging SystemValentina Magliocca, Maria Vinci, Tiziana Persichini, et al.
International Journal of Molecular Sciences|April 12, 2022
Modeling PCDH19-CE: From 2D Stem Cell Model to 3D Brain OrganoidsRossella Borghi, Valentina Magliocca, Marina Trivisano, et al.
Oncotarget|January 18, 2018
Identifying the dynamics of actin and tubulin polymerization in iPSCs and in iPSC-derived neuronsValentina Magliocca, Stefania Petrini, Tiziana Franchin, et al.
Journal of Clinical Medicine|July 2, 2021
Dissecting the Role of PCDH19 in Clustering Epilepsy by Exploiting Patient-Specific Models of NeurogenesisRossella Borghi, Valentina Magliocca, Stefania Petrini, et al.
International Journal of Molecular Sciences|May 13, 2023
CRISPR/Cas9 and piggyBac Transposon-Based Conversion of a Pathogenic Biallelic TBCD Variant in a Patient-Derived iPSC Line Allows Correction of PEBAT-Related EndophenotypesValentina Muto, Federica Benigni, Valentina Magliocca, et al.
Frontiers in Cellular Neuroscience|March 26, 2025
Riboflavin transporter deficiency: AAV9-SLC52A2 gene therapy as a new therapeutic strategyCecilia Mei, Valentina Magliocca, Xin Chen, et al.
Frontiers in Cellular Neuroscience|August 8, 2024
Modeling riboflavin transporter deficiency type 2: from iPSC-derived motoneurons to iPSC-derived astrocytesValentina Magliocca, Angela Lanciotti, Elena Ambrosini, et al.
International Journal of Molecular Sciences|October 10, 2020
Antioxidant Amelioration of Riboflavin Transporter Deficiency in Motoneurons Derived from Patient-Specific Induced Pluripotent Stem CellsChiara Marioli, Valentina Magliocca, Stefania Petrini, et al.
Archives of Biochemistry and Biophysics|November 24, 2025
Altered dimerization of certain riboflavin transporter 2 mutants: a possible source of UPR, altered calcium signalling and mitochondrial derangements in RTD2Maria Tolomeo, Valentina Magliocca, Stefania Petrini, et al.
Human Molecular Genetics|August 26, 2022
Dominantly acting KIF5B variants with pleiotropic cellular consequences cause variable clinical phenotypesElisabetta Flex, Shahad Albadri, Francesca Clementina Radio, et al.
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