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The Biochemical Journal|September 30, 2011
PDZ domains: the building blocks regulating tumorigenesisVanitha Krishna Subbaiah, Christian Kranjec, Miranda Thomas, et al.Virology|March 25, 2009
The high-risk HPV E6 oncoprotein preferentially targets phosphorylated nuclear forms of hDlgNisha Narayan, Vanitha Krishna Subbaiah, Lawrence BanksSmall Gtpases|June 21, 2011
The mechanism and implications of hScrib regulation of ERKKazunori Nagasaka, Paola Massimi, David Pim, et al.International Journal of Cancer|March 7, 2012
Regulation of the DLG tumor suppressor by β-cateninVanitha Krishna Subbaiah, Nisha Narayan, Paola Massimi, et al.Virology Journal|January 21, 2011
Analysis of the PDZ binding specificities of Influenza A virus NS1 proteinsMiranda Thomas, Christian Kranjec, Kazunori Nagasaka, et al.Plos One|January 30, 2013
A novel interaction between hScrib and PP1γ downregulates ERK signaling and suppresses oncogene-induced cell transformationKazunori Nagasaka, Takayuki Seiki, Aki Yamashita, et al.Journal of Virology|December 3, 2010
A systematic analysis of human papillomavirus (HPV) E6 PDZ substrates identifies MAGI-1 as a major target of HPV type 16 (HPV-16) and HPV-18 whose loss accompanies disruption of tight junctionsChristian Kranjec, Lawrence BanksJournal of Virology|April 4, 2014
Restoration of MAGI-1 expression in human papillomavirus-positive tumor cells induces cell growth arrest and apoptosisChristian Kranjec, Paola Massimi, Lawrence BanksPlos Pathogens|March 3, 2012
The invasive capacity of HPV transformed cells requires the hDlg-dependent enhancement of SGEF/RhoG activityVanitha Krishna Subbaiah, Paola Massimi, Siaw Shi Boon, et al.Journal of Molecular Biology|April 23, 2018
Upsetting the Balance: When Viruses Manipulate Cell Polarity ControlMiranda Thomas, Lawrence BanksPageof 19