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W J Neubert

Showing results (21-30 of 36) with videos related to

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Ear, Nose, & Throat Journal|September 2, 2000
Persistent measles virus infection as a possible cause of otosclerosis: state of the artH P Niedermeyer, W Arnold, W J Neubert, et al.
Advances in Oto-Rhino-Laryngology|January 25, 2007
Measles virus and otosclerosisH P Niedermeyer, T Gantumur, W J Neubert, et al.
Journal of Virology|March 1, 1991
Sendai virus protein-protein interactions studied by a protein-blotting protein-overlay technique: mapping of domains on NP protein required for binding to P proteinH E Homann, W Willenbrink, C J Buchholz, et al.
Virology|November 1, 1994
The carboxy-terminal domain of Sendai virus nucleocapsid protein is involved in complex formation between phosphoprotein and nucleocapsid-like particlesC J Buchholz, C Retzler, H E Homann, et al.
HNO|March 1, 1996
[Pathogenesis of otosclerosis. "State of the art"]W Arnold, H P Niedermeyer, H J Altermatt, et al.
Otolaryngologia Polska = the Polish Otolaryngology|August 5, 2000
Progress in basic research of otosclerosisW Arnold, R Sedlmeier, I Wiest, et al.
Journal of Virology|July 1, 1997
Sendai virus efficiently infects cells via the asialoglycoprotein receptor and requires the presence of cleaved F0 precursor proteins for this alternative route of cell entryM Bitzer, U Lauer, C Baumann, et al.
Journal of Virology|October 1, 1993
The conserved N-terminal region of Sendai virus nucleocapsid protein NP is required for nucleocapsid assemblyC J Buchholz, D Spehner, R Drillien, et al.
The Journal of General Virology|May 6, 1998
Sendai virus-like particles devoid of haemagglutinin-neuraminidase protein infect cells via the human asialoglycoprotein receptorS Leyrer, M Bitzer, U Lauer, et al.
Journal of Inflammation|January 1, 1995
Conserved high-affinity NF-kappa B binding site in the interferon regulatory factor-1 promoter is not occupied by NF-kappa B in vivo and is transcriptionally inactiveT Rein, R Schreck, W Willenbrink, et al.
Pageof 4

Showing results (21-30 of 36) with videos related to

Sort By:
Pageof 4
Ear, Nose, & Throat Journal|September 2, 2000
Persistent measles virus infection as a possible cause of otosclerosis: state of the artH P Niedermeyer, W Arnold, W J Neubert, et al.
Advances in Oto-Rhino-Laryngology|January 25, 2007
Measles virus and otosclerosisH P Niedermeyer, T Gantumur, W J Neubert, et al.
Journal of Virology|March 1, 1991
Sendai virus protein-protein interactions studied by a protein-blotting protein-overlay technique: mapping of domains on NP protein required for binding to P proteinH E Homann, W Willenbrink, C J Buchholz, et al.
Virology|November 1, 1994
The carboxy-terminal domain of Sendai virus nucleocapsid protein is involved in complex formation between phosphoprotein and nucleocapsid-like particlesC J Buchholz, C Retzler, H E Homann, et al.
HNO|March 1, 1996
[Pathogenesis of otosclerosis. "State of the art"]W Arnold, H P Niedermeyer, H J Altermatt, et al.
Otolaryngologia Polska = the Polish Otolaryngology|August 5, 2000
Progress in basic research of otosclerosisW Arnold, R Sedlmeier, I Wiest, et al.
Journal of Virology|July 1, 1997
Sendai virus efficiently infects cells via the asialoglycoprotein receptor and requires the presence of cleaved F0 precursor proteins for this alternative route of cell entryM Bitzer, U Lauer, C Baumann, et al.
Journal of Virology|October 1, 1993
The conserved N-terminal region of Sendai virus nucleocapsid protein NP is required for nucleocapsid assemblyC J Buchholz, D Spehner, R Drillien, et al.
The Journal of General Virology|May 6, 1998
Sendai virus-like particles devoid of haemagglutinin-neuraminidase protein infect cells via the human asialoglycoprotein receptorS Leyrer, M Bitzer, U Lauer, et al.
Journal of Inflammation|January 1, 1995
Conserved high-affinity NF-kappa B binding site in the interferon regulatory factor-1 promoter is not occupied by NF-kappa B in vivo and is transcriptionally inactiveT Rein, R Schreck, W Willenbrink, et al.
Pageof 4