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Ear, Nose, & Throat Journal
|
September 2, 2000
Persistent measles virus infection as a possible cause of otosclerosis: state of the art
H P Niedermeyer, W Arnold, W J Neubert, et al.
Advances in Oto-Rhino-Laryngology
|
January 25, 2007
Measles virus and otosclerosis
H P Niedermeyer, T Gantumur, W J Neubert, et al.
Journal of Virology
|
March 1, 1991
Sendai virus protein-protein interactions studied by a protein-blotting protein-overlay technique: mapping of domains on NP protein required for binding to P protein
H E Homann, W Willenbrink, C J Buchholz, et al.
Virology
|
November 1, 1994
The carboxy-terminal domain of Sendai virus nucleocapsid protein is involved in complex formation between phosphoprotein and nucleocapsid-like particles
C J Buchholz, C Retzler, H E Homann, et al.
HNO
|
March 1, 1996
[Pathogenesis of otosclerosis. "State of the art"]
W Arnold, H P Niedermeyer, H J Altermatt, et al.
Otolaryngologia Polska = the Polish Otolaryngology
|
August 5, 2000
Progress in basic research of otosclerosis
W Arnold, R Sedlmeier, I Wiest, et al.
Journal of Virology
|
July 1, 1997
Sendai virus efficiently infects cells via the asialoglycoprotein receptor and requires the presence of cleaved F0 precursor proteins for this alternative route of cell entry
M Bitzer, U Lauer, C Baumann, et al.
Journal of Virology
|
October 1, 1993
The conserved N-terminal region of Sendai virus nucleocapsid protein NP is required for nucleocapsid assembly
C J Buchholz, D Spehner, R Drillien, et al.
The Journal of General Virology
|
May 6, 1998
Sendai virus-like particles devoid of haemagglutinin-neuraminidase protein infect cells via the human asialoglycoprotein receptor
S Leyrer, M Bitzer, U Lauer, et al.
Journal of Inflammation
|
January 1, 1995
Conserved high-affinity NF-kappa B binding site in the interferon regulatory factor-1 promoter is not occupied by NF-kappa B in vivo and is transcriptionally inactive
T Rein, R Schreck, W Willenbrink, et al.
Page
of 4
Search research articles
Search
Showing results (21-30 of 36) with videos related to
Sort By:
Page
of 4
Ear, Nose, & Throat Journal
|
September 2, 2000
Persistent measles virus infection as a possible cause of otosclerosis: state of the art
H P Niedermeyer, W Arnold, W J Neubert, et al.
Advances in Oto-Rhino-Laryngology
|
January 25, 2007
Measles virus and otosclerosis
H P Niedermeyer, T Gantumur, W J Neubert, et al.
Journal of Virology
|
March 1, 1991
Sendai virus protein-protein interactions studied by a protein-blotting protein-overlay technique: mapping of domains on NP protein required for binding to P protein
H E Homann, W Willenbrink, C J Buchholz, et al.
Virology
|
November 1, 1994
The carboxy-terminal domain of Sendai virus nucleocapsid protein is involved in complex formation between phosphoprotein and nucleocapsid-like particles
C J Buchholz, C Retzler, H E Homann, et al.
HNO
|
March 1, 1996
[Pathogenesis of otosclerosis. "State of the art"]
W Arnold, H P Niedermeyer, H J Altermatt, et al.
Otolaryngologia Polska = the Polish Otolaryngology
|
August 5, 2000
Progress in basic research of otosclerosis
W Arnold, R Sedlmeier, I Wiest, et al.
Journal of Virology
|
July 1, 1997
Sendai virus efficiently infects cells via the asialoglycoprotein receptor and requires the presence of cleaved F0 precursor proteins for this alternative route of cell entry
M Bitzer, U Lauer, C Baumann, et al.
Journal of Virology
|
October 1, 1993
The conserved N-terminal region of Sendai virus nucleocapsid protein NP is required for nucleocapsid assembly
C J Buchholz, D Spehner, R Drillien, et al.
The Journal of General Virology
|
May 6, 1998
Sendai virus-like particles devoid of haemagglutinin-neuraminidase protein infect cells via the human asialoglycoprotein receptor
S Leyrer, M Bitzer, U Lauer, et al.
Journal of Inflammation
|
January 1, 1995
Conserved high-affinity NF-kappa B binding site in the interferon regulatory factor-1 promoter is not occupied by NF-kappa B in vivo and is transcriptionally inactive
T Rein, R Schreck, W Willenbrink, et al.
Page
of 4