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The Journal of Biological Chemistry|April 1, 1994
3,5,3'-L-triiodothyronine (thyroid hormone)-induced protein-DNA interactions in the thyroid hormone response elements and cell type-specific elements of the rat growth hormone gene revealed by in vivo dimethyl sulfate footprintingW R Force, S R SpindlerThe Journal of Biological Chemistry|January 10, 1998
Dominant negative mutants of TRAF3 reveal an important role for the coiled coil domains in cell death signaling by the lymphotoxin-beta receptorW R Force, T C Cheung, C F WareThe Journal of Biological Chemistry|March 25, 1994
Homodimer and heterodimer DNA binding and transcriptional responsiveness to triiodothyronine (T3) and 9-cis-retinoic acid are determined by the number and order of high affinity half-sites in a T3 response elementW R Force, J B Tillman, C N Sprung, et al.The Journal of Biological Chemistry|February 7, 2001
Discrete signaling regions in the lymphotoxin-beta receptor for tumor necrosis factor receptor-associated factor binding, subcellular localization, and activation of cell death and NF-kappaB pathwaysW R Force, A A Glass, C A Benedict, et al.Journal of Immunology (Baltimore, Md. : 1950)|December 1, 1995
Mouse lymphotoxin-beta receptor. Molecular genetics, ligand binding, and expressionW R Force, B N Walter, C Hession, et al.Proceedings of the National Academy of Sciences of the United States of America|March 18, 1997
Lymphotoxin-beta receptor signaling complex: role of tumor necrosis factor receptor-associated factor 3 recruitment in cell death and activation of nuclear factor kappaBT L VanArsdale, S L VanArsdale, W R Force, et al.Molecular Endocrinology (Baltimore, Md.)|September 1, 1992
Positive and negative thyroid hormone response elements are composed of strong and weak half-sites 10 nucleotides in lengthH S Kim, D E Crone, C N Sprung, et al.Nature|February 2, 1995
Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomasT Brunner, R J Mogil, D LaFace, et al.Pageof 1