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Neurology
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April 13, 2005
Evidence of increased oxidative damage in subjects with mild cognitive impairment
J N Keller, F A Schmitt, S W Scheff, et al.
Alzheimer Disease and Associated Disorders
|
January 1, 1992
Alzheimer disease brain extract stimulates branching of laminin-mediated neuronal processes
S D Kittur, H Endo, W H Adler, et al.
Neurodegeneration : a Journal for Neurodegenerative Disorders, Neuroprotection, and Neuroregeneration
|
December 1, 1995
Neutron activation analysis of trace elements in motor neuron disease spinal cord
W R Markesbery, W D Ehmann, J M Candy, et al.
Neurobiology of Aging
|
November 26, 2008
Prodromal clinical manifestations of neuropathologically confirmed Lewy body disease
G A Jicha, F A Schmitt, E Abner, et al.
Journal of Neuropathology and Experimental Neurology
|
May 22, 1998
Distribution of reducible 4-hydroxynonenal adduct immunoreactivity in Alzheimer disease is associated with APOE genotype
K S Montine, E Reich, M D Neely, et al.
Journal of Neuropathology and Experimental Neurology
|
May 26, 1998
N-terminal heterogeneity of parenchymal and cerebrovascular Abeta deposits
T L Tekirian, T C Saido, W R Markesbery, et al.
Neurology
|
October 26, 1999
Elevated CSF prostaglandin E2 levels in patients with probable AD
T J Montine, K R Sidell, B C Crews, et al.
Annals of Neurology
|
November 18, 1998
Protein modification by the lipid peroxidation product 4-hydroxynonenal in the spinal cords of amyotrophic lateral sclerosis patients
W A Pedersen, W Fu, J N Keller, et al.
Journal of Neurochemistry
|
July 20, 2001
The glial glutamate transporter, GLT-1, is oxidatively modified by 4-hydroxy-2-nonenal in the Alzheimer's disease brain: the role of Abeta1-42
C M Lauderback, J M Hackett, F F Huang, et al.
Biochemistry
|
May 1, 2001
Vulnerability of synaptosomes from apoE knock-out mice to structural and oxidative modifications induced by A beta(1-40): implications for Alzheimer's disease
C M Lauderback, J M Hackett, J N Keller, et al.
Page
of 26
Search research articles
Search
Showing results (211-220 of 253) with videos related to
Sort By:
Page
of 26
Neurology
|
April 13, 2005
Evidence of increased oxidative damage in subjects with mild cognitive impairment
J N Keller, F A Schmitt, S W Scheff, et al.
Alzheimer Disease and Associated Disorders
|
January 1, 1992
Alzheimer disease brain extract stimulates branching of laminin-mediated neuronal processes
S D Kittur, H Endo, W H Adler, et al.
Neurodegeneration : a Journal for Neurodegenerative Disorders, Neuroprotection, and Neuroregeneration
|
December 1, 1995
Neutron activation analysis of trace elements in motor neuron disease spinal cord
W R Markesbery, W D Ehmann, J M Candy, et al.
Neurobiology of Aging
|
November 26, 2008
Prodromal clinical manifestations of neuropathologically confirmed Lewy body disease
G A Jicha, F A Schmitt, E Abner, et al.
Journal of Neuropathology and Experimental Neurology
|
May 22, 1998
Distribution of reducible 4-hydroxynonenal adduct immunoreactivity in Alzheimer disease is associated with APOE genotype
K S Montine, E Reich, M D Neely, et al.
Journal of Neuropathology and Experimental Neurology
|
May 26, 1998
N-terminal heterogeneity of parenchymal and cerebrovascular Abeta deposits
T L Tekirian, T C Saido, W R Markesbery, et al.
Neurology
|
October 26, 1999
Elevated CSF prostaglandin E2 levels in patients with probable AD
T J Montine, K R Sidell, B C Crews, et al.
Annals of Neurology
|
November 18, 1998
Protein modification by the lipid peroxidation product 4-hydroxynonenal in the spinal cords of amyotrophic lateral sclerosis patients
W A Pedersen, W Fu, J N Keller, et al.
Journal of Neurochemistry
|
July 20, 2001
The glial glutamate transporter, GLT-1, is oxidatively modified by 4-hydroxy-2-nonenal in the Alzheimer's disease brain: the role of Abeta1-42
C M Lauderback, J M Hackett, F F Huang, et al.
Biochemistry
|
May 1, 2001
Vulnerability of synaptosomes from apoE knock-out mice to structural and oxidative modifications induced by A beta(1-40): implications for Alzheimer's disease
C M Lauderback, J M Hackett, J N Keller, et al.
Page
of 26