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Molecular Psychiatry|March 23, 2019
Sequential formation of different layers of dystrophic neurites in Alzheimer's brainsMd Golam Sharoar, Xiangyou Hu, Xin-Ming Ma, et al.
Neuroscience and Biobehavioral Reviews|February 11, 2010
RTN/Nogo in forming Alzheimer's neuritic plaquesMarguerite Prior, Qi Shi, Xiangyou Hu, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 24, 2009
Reduced amyloid deposition in mice overexpressing RTN3 is adversely affected by preformed dystrophic neuritesQi Shi, Marguerite Prior, Wanxia He, et al.
Scientific Reports|July 16, 2024
IRAK4 is an immunological checkpoint in neuropsychiatric systemic lupus erythematosusAntoine Ménoret, Federica Agliano, Timofey A Karginov, et al.
Stem Cell Reports|July 4, 2017
BACE1 Deficiency Causes Abnormal Neuronal Clustering in the Dentate GyrusHailong Hou, Qingyuan Fan, Wanxia He, et al.
Molecular Neurodegeneration|May 4, 2023
BACE1 regulates expression of Clusterin in astrocytes for enhancing clearance of β-amyloid peptidesJohn Zhou, Neeraj Singh, James Galske, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 15, 2013
Preventing formation of reticulon 3 immunoreactive dystrophic neurites improves cognitive function in miceQi Shi, Marguerite Prior, Xiangdong Zhou, et al.
Journal of Molecular Biology|September 19, 2006
Mapping of interaction domains mediating binding between BACE1 and RTN/Nogo proteinsWanxia He, Xiangyou Hu, Qi Shi, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|June 15, 2011
Interaction between amyloid precursor protein and Nogo receptors regulates amyloid depositionXiangdong Zhou, Xiangyou Hu, Wanxia He, et al.
Molecular Psychiatry|June 23, 2021
BACE1 controls synaptic function through modulating release of synaptic vesiclesBrati Das, Neeraj Singh, Annie Y Yao, et al.
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