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Oncogene|July 4, 1996
Bcl-2 blocks loss of mitochondrial membrane potential while ICE inhibitors act at a different step during inhibition of death induced by respiratory chain inhibitorsS Shimizu, Y Eguchi, W Kamiike, et al.The Journal of Biological Chemistry|December 16, 1998
Bcl-2 prevents caspase-independent cell deathS Okuno, S Shimizu, T Ito, et al.Oncogene|January 27, 2010
Involvement of JNK in the regulation of autophagic cell deathS Shimizu, A Konishi, Y Nishida, et al.Oncogene|May 16, 1996
Retardation of chemical hypoxia-induced necrotic cell death by Bcl-2 and ICE inhibitors: possible involvement of common mediators in apoptotic and necrotic signal transductionsS Shimizu, Y Eguchi, W Kamiike, et al.Proceedings of the National Academy of Sciences of the United States of America|December 9, 1998
Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondriaM Narita, S Shimizu, T Ito, et al.Nihon Hinyokika Gakkai Zasshi. the Japanese Journal of Urology|November 1, 1992
[Morphological and physiological change in the dis-used bladder of hemodialysis patients]Y TsujimotoProgress in Molecular and Subcellular Biology|January 1, 1996
bcl-2: antidote for cell deathY TsujimotoNihon Rinsho. Japanese Journal of Clinical Medicine|July 1, 1996
[Prevention of cell death by Bcl-2 family genes]Y TsujimotoProceedings of the National Academy of Sciences of the United States of America|March 1, 1989
Overexpression of the human BCL-2 gene product results in growth enhancement of Epstein-Barr virus-immortalized B cellsY TsujimotoJournal of Neural Transmission. Supplementum|December 29, 2000
Role of anti-apoptotic Bcl-2 protein in spinal muscular atrophyY TsujimotoPageof 153