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The Journal of Trauma|March 14, 1998
Prolonged immunodepression after trauma and hemorrhagic shockY X Xu, A Ayala, I H ChaudryShock (Augusta, Ga.)|April 3, 1999
Does Fas ligand or endotoxin contribute to thymic apoptosis during polymicrobial sepsis?A Ayala, Y X Xu, C S Chung, et al.The Journal of Surgical Research|August 12, 1998
Sepsis induces increased apoptosis in lamina propria mononuclear cells which is associated with altered cytokine gene expressionC S Chung, Y X Xu, I H Chaudry, et al.The Journal of Surgical Research|June 1, 1997
Mechanism of intestinal mucosal immune dysfunction following trauma-hemorrhage: increased apoptosis associated with elevated Fas expression in Peyer's patchesY X Xu, A Ayala, B Monfils, et al.Archives of Surgery (Chicago, Ill. : 1960)|November 20, 1998
Is Fas ligand or endotoxin responsible for mucosal lymphocyte apoptosis in sepsis?C S Chung, Y X Xu, W Wang, et al.The Journal of Surgical Research|February 15, 1997
Trauma-hemorrhage induces increased thymic apoptosis while decreasing IL-3 release and increasing GM-CSFY X Xu, M W Wichmann, A Ayala, et al.Shock (Augusta, Ga.)|January 1, 1996
Immune dysfunction in murine polymicrobial sepsis: mediators, macrophages, lymphocytes and apoptosisA Ayala, I H ChaudryNutrition (Burbank, Los Angeles County, Calif.)|January 1, 1995
Dietary n-3 polyunsaturated fatty acid modulation of immune cell function before or after traumaA Ayala, I H ChaudryAmerican Journal of Surgery|February 1, 1993
Mechanism of increased susceptibility to infection following hemorrhageI H Chaudry, A AyalaShock (Augusta, Ga.)|March 1, 1994
Hemorrhage induces a reduction in the capacity of macrophages to mobilize intracellular calcium secondary to formyl-methionyl-leucyl-phenylalanine stimulation: association with alterations in cells surface Fc receptor expression and increased prostaglandin releaseA Ayala, I H ChaudryPageof 94