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Nature Communications|October 4, 2014
Diurnal suppression of EGFR signalling by glucocorticoids and implications for tumour progression and treatmentMattia Lauriola, Yehoshua Enuka, Amit Zeisel, et al.Nature Genetics|December 13, 2016
SMARCB1-mediated SWI/SNF complex function is essential for enhancer regulationXiaofeng Wang, Ryan S Lee, Burak H Alver, et al.Cell|August 26, 2020
Large-Scale Topological Changes Restrain Malignant Progression in Colorectal CancerSarah E Johnstone, Alejandro Reyes, Yifeng Qi, et al.Nature Genetics|February 2, 2016
An oncogenic MYB feedback loop drives alternate cell fates in adenoid cystic carcinomaYotam Drier, Matthew J Cotton, Kaylyn E Williamson, et al.Nature|April 30, 2015
Th17 cells transdifferentiate into regulatory T cells during resolution of inflammationNicola Gagliani, Maria Carolina Amezcua Vesely, Andrea Iseppon, et al.Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|June 21, 2008
Stem cell-related "self-renewal" signature and high epidermal growth factor receptor expression associated with resistance to concomitant chemoradiotherapy in glioblastomaAnastasia Murat, Eugenia Migliavacca, Thierry Gorlia, et al.Nature Communications|September 27, 2019
c-Met activation leads to the establishment of a TGFβ-receptor regulatory network in bladder cancer progressionWen Jing Sim, Prasanna Vasudevan Iyengar, Dilraj Lama, et al.Nature Communications|November 10, 2019
Author Correction: c-Met activation leads to the establishment of a TGFβ-receptor regulatory network in bladder cancer progressionWen Jing Sim, Prasanna Vasudevan Iyengar, Dilraj Lama, et al.Cancer Discovery|April 10, 2020
Gain-of-Function Genetic Alterations of G9a Drive OncogenesisShinichiro Kato, Qing Yu Weng, Megan L Insco, et al.Blood|December 8, 2009
Down syndrome acute lymphoblastic leukemia, a highly heterogeneous disease in which aberrant expression of CRLF2 is associated with mutated JAK2: a report from the International BFM Study GroupLibi Hertzberg, Elena Vendramini, Ithamar Ganmore, et al.Pageof 14