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Frontiers in Cell and Developmental Biology|August 1, 2024
Ribosome heterogeneity in development and diseaseYuen Gao, Hongbing WangBiorxiv : the Preprint Server for Biology|August 7, 2023
Ribosome Heterogeneity in Development and DiseaseYuen Gao, Hongbing WangFrontiers in Behavioral Neuroscience|April 22, 2022
Neural Hyperactivity Is a Core Pathophysiological Change Induced by Deletion of a High Autism Risk Gene <i>Ash1L</i> in the Mouse BrainYuen Gao, Mohammad B Aljazi, Jin HeFrontiers in Behavioral Neuroscience|June 17, 2022
<i>Kdm6b</i> Haploinsufficiency Causes ASD/ADHD-Like Behavioral Deficits in MiceYuen Gao, Mohammad B Aljazi, Jin HeFrontiers in Pharmacology|September 13, 2023
A glimpse of the connection between PPARγ and macrophageLexiang Yu, Yuen Gao, Nicole Aaron, et al.Biomedicines|December 30, 2025
Pharmacological Interventions in Autism Spectrum Disorder: A Comprehensive Review of Mechanisms and EfficacyEva Sclabassi, Sophie Peret, Chunqi Qian, et al.Neuroscience Letters|September 12, 2021
Vorinostat, a histone deacetylase inhibitor, ameliorates the sociability and cognitive memory in an Ash1L-deletion-induced ASD/ID mouse modelYuen Gao, Mohammad B Aljazi, Yan Wu, et al.Biochemical and Biophysical Research Communications|February 19, 2022
SMYD5 is a histone H3-specific methyltransferase mediating mono-methylation of histone H3 lysine 36 and 37Mohammad B Aljazi, Yuen Gao, Yan Wu, et al.Iscience|October 26, 2020
Cell Signaling Coordinates Global PRC2 Recruitment and Developmental Gene Expression in Murine Embryonic Stem CellsMohammad B Aljazi, Yuen Gao, Yan Wu, et al.Frontiers in Oncology|October 25, 2021
Histone H3K36me2-Specific Methyltransferase ASH1L Promotes MLL-AF9-Induced LeukemogenesisMohammad B Aljazi, Yuen Gao, Yan Wu, et al.Pageof 3