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Annals of the New York Academy of Sciences|July 24, 2010
Bioenergetics and cell deathYulia Kushnareva, Donald D NewmeyerPlos Biology|October 11, 2012
Bax activation initiates the assembly of a multimeric catalyst that facilitates Bax pore formation in mitochondrial outer membranesYulia Kushnareva, Alexander Y Andreyev, Tomomi Kuwana, et al.Molecular Biology of the Cell|October 2, 2009
Caspase-independent mitochondrial cell death results from loss of respiration, not cytotoxic protein releaseLydia Lartigue, Yulia Kushnareva, Youngmo Seong, et al.The Journal of Biological Chemistry|March 19, 2022
Disruption of mitochondrial quality control genes promotes caspase-resistant cell survival following apoptotic stimuliYulia Kushnareva, Vivian Moraes, Julian Suess, et al.Molecular Cell|August 12, 2008
Opa1-mediated cristae opening is Bax/Bak and BH3 dependent, required for apoptosis, and independent of Bak oligomerizationRyuji Yamaguchi, Lydia Lartigue, Guy Perkins, et al.Current Opinion in Cell Biology|December 4, 2003
Bcl-2-family proteins and the role of mitochondria in apoptosisTomomi Kuwana, Donald D NewmeyerCell|February 26, 2003
Mitochondria: releasing power for life and unleashing the machineries of deathDonald D Newmeyer, Shelagh Ferguson-MillerThe Biochemical Journal|August 16, 2002
Complex I-mediated reactive oxygen species generation: modulation by cytochrome c and NAD(P)+ oxidation-reduction stateYulia Kushnareva, Anne N Murphy, Alexander AndreyevMolecular Cell|December 9, 2014
Mitochondrial shape governs BAX-induced membrane permeabilization and apoptosisThibaud T Renault, Konstantinos V Floros, Rana Elkholi, et al.The Journal of Clinical Investigation|October 4, 2005
Mitochondria: pharmacological manipulation of cell deathLisa Bouchier-Hayes, Lydia Lartigue, Donald D NewmeyerPageof 4