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Apoptosis : an International Journal on Programmed Cell Death|August 18, 2004
Fatty acid protection from palmitic acid-induced apoptosis is lost following PI3-kinase inhibitionN Beeharry, J A Chambers, I C GreenApoptosis : an International Journal on Programmed Cell Death|August 18, 2004
Integrin and cytoskeleton behaviour in human neuroblastoma cells during hyperthermia-related apoptosisF Luchetti, F Mannello, B Canonico, et al.Apoptosis : an International Journal on Programmed Cell Death|August 18, 2004
Vulnerability of mouse cortical neurons to doxorubicin-induced apoptosis is strain-dependent and is correlated with mRNAs encoding Fas, Fas-Ligand, and metalloproteinasesM Wetzel, J Tibbitts, G A Rosenberg, et al.Apoptosis : an International Journal on Programmed Cell Death|July 20, 2004
Programmed cell death in the development of the vertebrate inner earY León, S Sánchez-Galiano, I GorospeApoptosis : an International Journal on Programmed Cell Death|July 20, 2004
Id-1 expression and cell survivalY-C Wong, X Wang, M-T LingApoptosis : an International Journal on Programmed Cell Death|July 20, 2004
Targeting melanoma inhibitor of apoptosis protein with cancer immunotherapyJ C Schmollinger, G DranoffApoptosis : an International Journal on Programmed Cell Death|July 20, 2004
Mitochondrial translocation of p53 and mitochondrial membrane potential (Delta Psi m) dissipation are early events in staurosporine-induced apoptosis of wild type and mutated p53 epithelial cellsJ F Charlot, J L Prétet, C Haughey, et al.Apoptosis : an International Journal on Programmed Cell Death|July 20, 2004
Differentiation-induced HL-60 cell apoptosis: a mechanism independent of mitochondrial disruption?B T Doyle, A J O'Neill, J M Fitzpatrick, et al.Apoptosis : an International Journal on Programmed Cell Death|June 12, 2004
Caspase-9 regulation: an updateC R Johnson, W D JarvisApoptosis : an International Journal on Programmed Cell Death|June 12, 2004
Apoptosis induction by the natural product cancer chemopreventive agent deguelin is mediated through the inhibition of mitochondrial bioenergeticsN Hail, R LotanPageof 314