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Molecular Endocrinology (Baltimore, Md.)|January 15, 2005
Elevated glucose attenuates human insulin gene promoter activity in INS-1 pancreatic beta-cells via reduced nuclear factor binding to the A5/core and Z elementMaria F Pino, Diana Z Ye, Katrina D Linning, et al.Molecular Endocrinology (Baltimore, Md.)|December 14, 2004
Ligand-independent actions of the vitamin D receptor maintain hair follicle homeostasisKristi Skorija, Megan Cox, Jeanne M Sisk, et al.Molecular Endocrinology (Baltimore, Md.)|December 14, 2004
11beta-Hydroxysteroid dehydrogenase expression and glucocorticoid synthesis are directed by a molecular switch during osteoblast differentiationM Eijken, M Hewison, M S Cooper, et al.Molecular Endocrinology (Baltimore, Md.)|December 18, 2004
Relaxin stimulates protein kinase C zeta translocation: requirement for cyclic adenosine 3',5'-monophosphate productionBao T Nguyen, Carmen W DessauerMolecular Endocrinology (Baltimore, Md.)|December 25, 2004
Novel association of Vav2 and Nek3 modulates signaling through the human prolactin receptorSommer L Miller, Jamie E DeMaria, David O Freier, et al.Molecular Endocrinology (Baltimore, Md.)|December 25, 2004
Targeted knockdown of insulin-like growth factor binding protein-2 disrupts cardiovascular development in zebrafish embryosAntony W Wood, Peter J Schlueter, Cunming DuanMolecular Endocrinology (Baltimore, Md.)|February 1, 1992
H-2RIIBP expressed from a baculovirus vector binds to multiple hormone response elementsM S Marks, B Z Levi, J H Segars, et al.Molecular Endocrinology (Baltimore, Md.)|January 8, 2005
Beta-arrestin- and G protein receptor kinase-mediated calcium-sensing receptor desensitizationMin Pi, Robert H Oakley, Diane Gesty-Palmer, et al.Molecular Endocrinology (Baltimore, Md.)|February 12, 2005
Transcriptional changes underlying the secretory activation phase of mammary gland developmentMatthew J Naylor, Samantha R Oakes, Margaret Gardiner-Garden, et al.Molecular Endocrinology (Baltimore, Md.)|February 8, 2005
The nuclear receptor corepressor deacetylase activating domain is essential for repression by thyroid hormone receptorTakahiro Ishizuka, Mitchell A LazarPageof 558