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Hypercalcemia in a patient with tuberculous mediastinal lymphadenopathy

T T Tan1, B C Lee, B M Zainuddin

  • 1Department of Medicine, University Kebangsaan Malaysia, Kuala Lumpur.

Singapore Medical Journal
|February 1, 1991
PubMed

Insights

Tuberculosis can cause hypercalcemia, a condition of high blood calcium. This case study shows that treating tuberculosis effectively resolves hypercalcemia, highlighting the importance of considering this link in diagnosis.

Area of Science:

  • Internal Medicine
  • Pulmonology
  • Endocrinology

Background:

  • Hypercalcemia, a condition characterized by elevated serum calcium levels, can manifest with diverse symptoms and etiologies.
  • Tuberculosis (TB), a significant global infectious disease, has been anecdotally linked to hypercalcemia, particularly in Western populations.
  • Limited local data exists regarding the prevalence and management of hypercalcemia associated with tuberculosis in Indian patients.

Purpose of the Study:

  • To report a case of hypercalcemia secondary to tuberculosis in a 48-year-old Indian female.
  • To highlight the diagnostic approach and successful management of this rare clinical presentation.
  • To contribute to the understanding of tuberculosis-associated hypercalcemia in a local context.

Main Methods:

  • Clinical presentation review of a patient with hypercalcemia symptoms.
  • Diagnostic imaging including chest radiography to assess for pulmonary abnormalities.
  • Histopathological examination of enlarged hilar lymph nodes for tuberculosis confirmation.
  • Monitoring of serum calcium levels during anti-tuberculous therapy.

Main Results:

  • The patient presented with symptoms consistent with hypercalcemia.
  • Chest radiographs revealed bilateral hilar lymphadenopathy with normal lung fields.
  • Histological analysis of hilar lymph node biopsy confirmed tuberculosis.
  • Hypercalcemia resolved completely after one month of anti-tuberculous treatment.

Conclusions:

  • Tuberculosis should be considered in the differential diagnosis of hypercalcemia, even in the absence of typical pulmonary findings.
  • Effective anti-tuberculous treatment is crucial for resolving hypercalcemia of this etiology.
  • Further local studies are warranted to elucidate the prevalence and underlying mechanisms of tuberculosis-associated hypercalcemia.

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