Videos de Experimentos Relacionados
Patogénesis del VIH: Nef pierde el control de su patogénesis.
John L Foster1, J Victor Garcia
1Division of Infectious Diseases, Department of Internal Medicine, University of Texas Southwestern Medical Center at Dallas, 75390, USA.
Cell
|June 17, 2006
Resumen
El virus de inmunodeficiencia simio (SIV) proteína Nef regula a la baja los receptores de células T, a diferencia del VIH-1 Nef. Esta diferencia funcional puede explicar por qué el VIH-1 es más patógeno.
Videos de Conceptos Relacionados
También podría leer
Artículos Relacionados
Artículos vinculados a este trabajo por autores compartidos, revista y gráfico de citas.
Ordenar por
Same author
The decay of HIV under anti-retroviral therapy is biphasic even in humanized mice with just T cells.
Journal of virology·2025
Same author
Combining spironolactone to antiretroviral therapy accelerates HIV decay in humanized mice.
Emerging microbes & infections·2025
Same author
SARS-CoV-2 infection induces pro-fibrotic and pro-thrombotic foam cell formation.
Nature microbiology·2025
Same author
Envelope-dimer epitope 1 (EDE1) antibody (C10) treatment significantly reduces Zika virus replication in the male and female reproductive tracts.
Journal of virology·2025
Same author
Humanized Mouse Systems to Study Viral Infection: A New Era in Immunology Research.
Annual review of immunology·2025
Same author
RSV infection of humanized lung-only mice induces pathological changes resembling severe bronchiolitis and bronchopneumonia.
Frontiers in virology (Lausanne, Switzerland)·2024
Same journal
Electromagnetic field-inducible in vivo gene switch for remote spatiotemporal control of gene expression.
Cell·2026
Same journal
Iron drives protease-independent cleavage of gasdermin D in allergic airway diseases.
Cell·2026