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Aron Gyorgypal1, Robert M Anthony1
1Center for Immunology and Inflammatory Diseases, Division of Rheumatology, Allergy and Immunology, Department of Medicine, Massachusetts General Hospital, Harvard Medical School, Boston, MA, USA.
癌症干细胞中的TP53突变驱动Interleukin-34 (IL-34) 释放,它重编程巨细胞以抑制T细胞反应并促进瘤免疫逃避.
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