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[Amiodarone induced retrobulbar neuritis. Clinical case]

J Bartolucci1, P Ibáñez, C Verdugo

  • 1Departamento de Cardiología, Hospital Dr. Gustavo Fricke, Facultad de Medicina, Universidad de Valparaíso, Chile. bart1-kong2@entelchile.net

Revista Medica De Chile
|February 11, 2000
PubMed

Insights

This case report highlights a potential adverse effect of amiodarone, an antiarrhythmic drug. The patient experienced vision changes, including visual field loss, after treatment, suggesting amiodarone-induced optic neuropathy.

Area of Science:

  • Ophthalmology
  • Cardiology
  • Pharmacology

Background:

  • A 66-year-old male with coronary heart disease and long sightedness presented with paroxysmal atrial fibrillation.
  • Treatment involved intravenous amiodarone at a total dose of 6g over 72 hours.

Observation:

  • The patient initially noted improved long sightedness after three days of amiodarone treatment.
  • Subsequently, blurred vision, frontal headache, and visual field defects (concentric retraction, centrocecal scotoma) developed.

Findings:

  • Amiodarone was discontinued, and dexamethasone treatment was initiated.
  • Visual function and visual fields normalized within three days of amiodarone withdrawal.

Implications:

  • This case suggests amiodarone may be a potential cause of retrobulbar optic neuritis.
  • Further investigation is warranted to establish a causal link between amiodarone and optic neuropathy.

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