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Superparamagnetic iron oxide-enhanced MRI of atherosclerotic plaques in Watanabe hereditable hyperlipidemic rabbits

S A Schmitz1, S E Coupland, R Gust

  • 1Department of Radiology and Nuclear Medicine, Universitätsklinikum Benjamin Franklin, Berlin, Germany. s.schmitz@medizin.fu-berlin.de

Investigative Radiology
|August 18, 2000
PubMed
Abstract

Insights

Superparamagnetic iron oxide (SPIO) accumulates in atherosclerotic plaques, detectable by MRI. This finding suggests SPIO

Area of Science:

  • Biomedical Imaging
  • Cardiovascular Research
  • Nanotechnology

Background:

  • Inflammatory atherosclerotic plaques exhibit increased endothelial permeability and macrophage infiltration.
  • Superparamagnetic iron oxide (SPIO) nanoparticles target the monocyte-macrophage system.
  • SPIO may serve as a contrast agent for labeling inflammatory plaques.

Purpose of the Study:

  • To investigate SPIO uptake in atherosclerotic plaques in rabbits using MRI.
  • To correlate MRI findings with histological evidence of SPIO deposition.

Main Methods:

  • Watanabe hereditable hyperlipidemic rabbits underwent MRI at 1.5 T using a T2*-weighted sequence.
  • Animals were divided into four groups, with three receiving varying doses and timings of SPIO (25 nm carboxydextran-coated) and one control group.
  • In vivo MRI was compared with ex vivo histological iron staining.

Main Results:

  • A dose of 200 micromol Fe/kg SPIO resulted in focal signal loss in the aortic wall in 31% of images, significantly different from controls (P = 0.009).
  • Microscopy confirmed SPIO-iron within endothelial cells and macrophages in atherosclerotic lesions.
  • Lesions with SPIO uptake showed a high macrophage content.

Conclusions:

  • SPIO effectively accumulates in the aortic plaques of atherosclerotic rabbits.
  • The MRI signal loss is a characteristic finding of SPIO deposition in plaques.
  • SPIO's accumulation in plaques with high macrophage content and increased permeability suggests its potential for noninvasive assessment of inflammatory atherosclerotic plaques.

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