Immunology taught by Darwin

Same author

Human Immunodeficiency Virus Infection Impairs Th1 and Th17 Mycobacterium tuberculosis-Specific T-Cell Responses.

The Journal of infectious diseases·2018

Same author

TLR signaling in human antigen-presenting cells regulates MR1-dependent activation of MAIT cells.

European journal of immunology·2016

Same author

LLT1 and CD161 Expression in Human Germinal Centers Promotes B Cell Activation and CXCR4 Downregulation.

Journal of immunology (Baltimore, Md. : 1950)·2016

Same author

Immunological biomarkers predict HIV-1 viral rebound after treatment interruption.

Nature communications·2015

Same author

Molecular Analyses Define Vα7.2-Jα33+ MAIT Cell Depletion in HIV Infection: A Case-Control Study.

Medicine·2015

Same author

Structured observations reveal slow HIV-1 CTL escape.

PLoS genetics·2015

Same journal

Editorial Expression of Concern: Recognition of the nonclassical MHC class I molecule H2-M3 by the receptor Ly49A regulates the licensing and activation of NK cells.

Nature immunology·2026

Same journal

Inflammatory immune modulators of AML lung infiltration and respiratory failure.

Nature immunology·2026

Same journal

The neuroimmune system and cognition.

Nature immunology·2026

Same journal

Critical connections.

Nature immunology·2026

Same journal

Innate immune signaling and functions in astrocytes.

Nature immunology·2026

Same journal

Epigenetic mechanisms of inflammatory memory in the central nervous system.

Nature immunology·2026

Related Experiment Videos

Immunology taught by Darwin

Related Concept Videos

Human Immunodeficiency Virus Infection Impairs Th1 and Th17 Mycobacterium tuberculosis-Specific T-Cell Responses.

TLR signaling in human antigen-presenting cells regulates MR1-dependent activation of MAIT cells.

LLT1 and CD161 Expression in Human Germinal Centers Promotes B Cell Activation and CXCR4 Downregulation.

Immunological biomarkers predict HIV-1 viral rebound after treatment interruption.

Molecular Analyses Define Vα7.2-Jα33+ MAIT Cell Depletion in HIV Infection: A Case-Control Study.

Structured observations reveal slow HIV-1 CTL escape.

Editorial Expression of Concern: Recognition of the nonclassical MHC class I molecule H2-M3 by the receptor Ly49A regulates the licensing and activation of NK cells.

Inflammatory immune modulators of AML lung infiltration and respiratory failure.

The neuroimmune system and cognition.

Critical connections.

Innate immune signaling and functions in astrocytes.

Epigenetic mechanisms of inflammatory memory in the central nervous system.

Related Experiment Videos

Immunology taught by Darwin

Related Concept Videos

Related Articles

Human Immunodeficiency Virus Infection Impairs Th1 and Th17 Mycobacterium tuberculosis-Specific T-Cell Responses.

TLR signaling in human antigen-presenting cells regulates MR1-dependent activation of MAIT cells.

LLT1 and CD161 Expression in Human Germinal Centers Promotes B Cell Activation and CXCR4 Downregulation.

Immunological biomarkers predict HIV-1 viral rebound after treatment interruption.

Molecular Analyses Define Vα7.2-Jα33+ MAIT Cell Depletion in HIV Infection: A Case-Control Study.

Structured observations reveal slow HIV-1 CTL escape.

Editorial Expression of Concern: Recognition of the nonclassical MHC class I molecule H2-M3 by the receptor Ly49A regulates the licensing and activation of NK cells.

Inflammatory immune modulators of AML lung infiltration and respiratory failure.

The neuroimmune system and cognition.

Critical connections.

Innate immune signaling and functions in astrocytes.

Epigenetic mechanisms of inflammatory memory in the central nervous system.