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Published on: August 25, 2023
Left ventricular outflow tract obstruction due to anomalous insertion of papillary muscle
Tetsuya Nomura1, Yoshiaki Harada, Yoko Suzaki
1Department of Cardiology, Maizuru Medical Center, Yukinaga, Maizuru, Japan. prinom@eos.ocn.ne.jp
Insights
Anomalous papillary muscle in the heart can cause left ventricular outflow tract (LVOT) obstruction, leading to serious symptoms. Surgical correction of this rare condition effectively resolved the obstruction and associated complications.
Area of Science:
- Cardiology
- Cardiac Surgery
- Medical Imaging
Background:
- Left ventricular outflow tract (LVOT) obstruction can arise from various causes, including rare anatomical abnormalities.
- Accessory papillary muscles can abnormally encroach upon the LVOT, potentially leading to hemodynamic compromise.
Observation:
- A 56-year-old man presented with quadrantic hemianopsia, attributed to a cerebral embolism likely secondary to arrhythmia.
- Echocardiography revealed a hypertrophied anterolateral papillary muscle causing direct continuity with the anterior mitral leaflet, narrowing the LVOT.
- The LVOT obstruction resulted in intra-LV pressure overload and concentric left ventricular hypertrophy.
Findings:
- Cerebral angiography excluded organic stenosis as the cause of hemianopsia.
- Surgical intervention included mitral valve replacement, septal myectomy, and myectomy of the abnormal papillary muscle.
- Complete release of the LVOT obstruction was achieved, resolving the patient's condition.
Implications:
- Anomalous papillary muscle insertion is a rare but significant cause of LVOT obstruction.
- Echocardiography is a valuable diagnostic tool for identifying papillary muscle malformations.
- Surgical management of this anomaly can be curative, preventing further embolic events and hemodynamic dysfunction.
Abstract:
A 56-year-old man who complained of quadrantic hemianopsia was admitted to determine its etiology. Cerebral angiography revealed no organic stenosis. Echocardiography showed clear direct continuity between a hypertrophied anterolateral papillary muscle and the anterior mitral leaflet, and the left ventricular (LV) outflow tract (LVOT) was narrowed by the presence of an accessory papillary muscle. The LVOT obstruction caused an intra-LV pressure overload that resulted in LV concentric hypertrophy. Arrhythmia, such as paroxysmal atrial fibrillation (PAF), was thought to have caused a cerebral embolism. Mitral valve replacement (MVR), septal myectomy, and myectomy of the abnormal papillary muscle were performed, and complete release of the LVOT obstruction was accomplished. Anomalous insertion of papillary muscle is a rare cause of LVOT obstruction. Echocardiography was useful in identifying the papillary muscle malformation, and surgery was completely curative.
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