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[Mumps infection and disorders of glucose tolerance]

J Jovanović1, E Dimić, K Knezević

  • 1Klinika za infektivne bolesti, Medicinski fakultet Novi Sad.

Medicinski Pregled
|January 1, 1991
PubMed

Insights

Mumps virus infection can cause juvenile diabetes mellitus by directly affecting pancreatic beta cells. These glucose tolerance disturbances persist long-term and are not linked to hereditary factors or exocrine pancreas changes.

Area of Science:

  • Virology
  • Endocrinology
  • Immunology

Context:

  • Juvenile diabetes mellitus (type 1 diabetes) has a suspected viral etiology.
  • Mumps virus is frequently implicated as a potential cause.
  • High incidence of diabetes and mumps outbreaks in the region necessitated this study.

Purpose:

  • To investigate the influence of mumps infection on glucose tolerance.
  • To determine the direct impact of mumps virus on pancreatic beta cells.
  • To explore the long-term effects of mumps on carbohydrate metabolism.

Summary:

  • Mumps infection leads to carbohydrate metabolism disturbances, affecting glucose tolerance.
  • These disturbances occur due to the direct influence of the mumps virus on pancreatic beta cells.
  • Effects on glucose tolerance persist for at least two years post-infection, independent of hereditary factors or exocrine pancreas changes.

Impact:

  • Provides evidence for mumps virus as an etiological factor in juvenile diabetes.
  • Highlights the direct role of viral infection in pancreatic beta cell dysfunction.
  • Suggests "slow virus infection" mechanisms may contribute to glucose intolerance.

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