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Related Concept Videos

Pulmonary Embolism I: Introduction01:29

Pulmonary Embolism I: Introduction

Pulmonary embolism (PE) occurs when a thrombus, fat or air embolus, amniotic fluid, or tumor tissue blocks one or more pulmonary arteries. These blockages originate in the venous system or the right side of the heart.EtiologyPE primarily arises from deep vein thrombosis (DVT) and other hypercoagulable states, such as inherited thrombophilias. Additional etiological factors include venous stasis, commonly seen in obesity, and endothelial injury from surgery and trauma. Less common causes include...
Pulmonary Embolism I: Introduction01:19

Pulmonary Embolism I: Introduction

A blood clot, or thrombus, is a semi-solid mass composed of fibrin, platelets, and red blood cells. When it forms within a vessel, it can obstruct blood flow, known as thrombosis. If part of the clot detaches, it becomes an embolus that can travel and block distant vessels. When this occurs in the pulmonary arteries, it causes a condition known as pulmonary embolism (PE).Origin and ImpactMost often, the embolus originates from a thrombus in the deep veins of the lower limbs, a condition called...
Pulmonary Embolism II: Diagnostic Studies and Interprofessional Care01:29

Pulmonary Embolism II: Diagnostic Studies and Interprofessional Care

Diagnosing Pulmonary EmbolismDiagnosing pulmonary embolism (PE) involves clinical assessment and advanced imaging tests. The preferred diagnostic tool is the spiral (helical) CT scan or CT angiography (CTA), which uses intravenous contrast media to visualize the pulmonary vasculature and identify emboli.A ventilation-perfusion (V/Q) scan is an alternative for patients unable to receive contrast media. This scan includes both perfusion and ventilation scanning. Perfusion scanning involves...
Pulmonary Embolism III: Nursing Management01:27

Pulmonary Embolism III: Nursing Management

A pulmonary embolism occurs when a thrombus, amniotic fluid, tumor tissue, fat, or air embolus blocks one or more pulmonary arteries. Effective nursing management and patient education are crucial for improving outcomes and preventing recurrence.Nursing management starts with obtaining a comprehensive patient history, particularly noting any history of deep vein thrombosis (DVT). Assess for clinical manifestations, including dyspnea, chest pain, crackles, heart murmurs, and signs of right-sided...
Pneumonia III: Complications and Assessment01:30

Pneumonia III: Complications and Assessment

Pneumonia poses the potential for numerous complications that warrant consideration. These complications include the following:
Pneumonia II: Pathophysiology01:29

Pneumonia II: Pathophysiology

The pathophysiology of pneumonia involves the following steps:

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Updated: Jun 27, 2026

A Porcine Model of Acute Autologous Pulmonary Embolism
07:44

A Porcine Model of Acute Autologous Pulmonary Embolism

Published on: September 6, 2024

[Septic pulmonary embolism - case report].

Luís Coentrão1, Jorge Oliveira

  • 1Internato Complementar de Nefrologia, Hospital S. João, Porto, Serviço de Medicina Interna. luis_coentrao@yahoo.com

Revista Portuguesa De Pneumologia
|November 22, 2008
PubMed
Summary
This summary is machine-generated.

Septic pulmonary embolism (SPE), often linked to intravenous drug use, presents unique diagnostic challenges. This case highlights SPE associated with tricuspid valve endocarditis, emphasizing the need for updated diagnostic criteria.

Related Experiment Videos

Last Updated: Jun 27, 2026

A Porcine Model of Acute Autologous Pulmonary Embolism
07:44

A Porcine Model of Acute Autologous Pulmonary Embolism

Published on: September 6, 2024

Area of Science:

  • Infectious Diseases
  • Cardiology
  • Pulmonology

Context:

  • Septic pulmonary embolism (SPE) is a clinical presentation of various disorders, frequently misdiagnosed.
  • Historically associated with intravenous drug abuse, its epidemiology is evolving.
  • Tricuspid valve endocarditis is a significant cause of SPE.

Purpose:

  • To report a case of SPE secondary to tricuspid valve endocarditis in an intravenous drug user.
  • To review the literature on SPE and propose diagnostic criteria.
  • To highlight diagnostic challenges and successful treatment strategies.

Summary:

  • A patient with a history of intravenous drug abuse presented with symptoms suggestive of pneumonia but was diagnosed with SPE and tricuspid valve endocarditis.
  • Blood cultures identified methicillin-susceptible Staphylococcus aureus, and echocardiography confirmed a tricuspid valve vegetation.
  • Targeted antimicrobial therapy led to successful treatment, underscoring the importance of accurate diagnosis and management.

Impact:

  • This case contributes to understanding the changing epidemiology of SPE.
  • The proposed diagnostic criteria aim to improve early and accurate diagnosis of SPE.
  • Effective management strategies for SPE associated with endocarditis can be refined based on this case and literature review.