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Drug-Induced Sleep Endoscopy (DISE) with Target Controlled Infusion (TCI) and Bispectral Analysis in Obstructive Sleep Apnea
Published on: December 6, 2016
[Correlation between clinic and polysomnographic findings in children with obstructive sleep apnea hypopnea syndrome]
Jiancong Huang1, Jin Ye, Qintai Yang
1Department of Otorhinolaryngology Head and Neck Surgery, the Third Affiliated Hospital of SunYat-sen University, Guangzhou, 510630, China.
Insights
Clinical diagnosis of obstructive sleep apnea hypopnea syndrome (OSAHS) in children does not reliably correlate with polysomnographic findings. Adenoid and tonsil hypertrophy did not predict apnea severity in this study.
Area of Science:
- Pediatric Sleep Medicine
- Otolaryngology
- Respiratory Medicine
Context:
- Obstructive sleep apnea hypopnea syndrome (OSAHS) is common in children.
- Clinical diagnosis often relies on symptoms and physical examination, including assessment of adenoid and tonsillar hypertrophy.
- Polysomnography (PSG) is the gold standard for diagnosing OSAHS.
Purpose:
- To investigate the correlation between polysomnographic findings and the degree of airway obstruction from adenoid and tonsillar hypertrophy in children with suspected OSAHS.
- To determine if clinical assessment of hypertrophy aligns with PSG-documented OSAHS severity.
Summary:
- This retrospective study analyzed 94 children clinically diagnosed with OSAHS who underwent PSG and endoscopy.
- A significant portion (38.3%) of children with clinical OSAHS had normal PSG findings.
- No correlation was found between the degree of tonsillar and/or adenoid hypertrophy and the severity of apnea on PSG.
Impact:
- Highlights a potential disconnect between clinical suspicion and objective PSG findings in pediatric OSAHS.
- Suggests that clinical evaluation of adenoid and tonsillar hypertrophy alone may not accurately predict OSAHS severity.
- Informs diagnostic approaches and the interpretation of clinical findings in pediatric sleep-disordered breathing.
Objective:
To evaluate the correlation between the polysomnographic findings and the degree of obstruction caused by adenoid and tonsillar hypertrophy in children with clinical history of apnea.
Method:
Retrospectively studied the children who were diagnosed clinically of, obstructive sleep apnea hypopnea syndrome (OSAHS) and underwent polysomnography and endoscopy. Patients were divided to OSAHS and non-OSAHS group according to polysomnographic findings.
Result:
Ninety-four children were involved in the study population, and 63 children of them were male. The mean age of the children at the time of inclusion in the study was 6.7 years. 36 children (38.3%) diagnosed OSAHS clinically had normal polysomnographic findings. No differences were found between children with PSG-documented OSAHS and others. Tonsillar and/or adenoid hypertrophy were not correlated to more severe apnea among enrolled children.
Conclusion:
There was no significant correlation between polysomnographic and clinical findings in children with OSAHS.
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