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Updated: May 14, 2026

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Published on: April 17, 2020
Modern outcomes of oesophageal atresia: single centre experience over the last twenty years
Antti I Koivusalo1, Mikko P Pakarinen, Risto J Rintala
1Section of Paediatric Surgery, Children's Hospital University of Helsinki, Finland. antti.koivusalo@hus.fi
Insights
This study shows that modern treatment for oesophageal atresia (OA) results in very low infant mortality and good esophageal function, allowing most children to eat orally. Long-gap OA, however, predicts complications.
Area of Science:
- Pediatric Surgery
- Congenital Abnormalities
- Gastroenterology
Background:
- Oesophageal atresia (OA) is a congenital condition requiring surgical intervention.
- Understanding contemporary outcomes is crucial for improving patient management.
- Long-term functional results and survival rates inform surgical strategies.
Purpose of the Study:
- To assess current outcomes for infants with oesophageal atresia (OA).
- To evaluate surgical repair success and long-term functional status.
- To identify predictors of complications following OA repair.
Main Methods:
- Retrospective review of hospital records for 130 infants with OA.
- Surgical repair methods included primary anastomosis, delayed anastomosis, and oesophageal replacement.
- Follow-up data analyzed for mortality, oral feeding achievement, and complications.
Main Results:
- Overall mortality was low at 2% (3/130).
- Oesophageal continuity was achieved in all patients, with 94% achieving oral feeds.
- Long-gap OA was identified as a significant predictor of post-repair complications.
Conclusions:
- Modern surgical management of OA yields excellent survival rates.
- Most infants achieve satisfactory oesophageal function and can feed orally.
- Early identification and management of long-gap OA are essential.
Aim Of The Study:
The aim of this study was assessment of the contemporary outcomes of oesophageal atresia (OA) in a national paediatric surgical centre.
Patients And Methods:
A review of the hospital records of all patients who underwent repair of oesophageal atresia (OA) in our institute between 1991 and 2011 was performed.
Results:
The study included 130 consecutive infants with OA: types A (n=4, 3%), B (n=2, 2%), C (n=110, 85%), D (n=5, 4%), and E (n=9, 7%). Median follow-up was 8.8 (range 0.1-21) years. Twenty-nine (22%) infants had cardiac and 76 (58%) other anomalies, and seventeen (13%) had a long-gap OA. The final repair was primary (n =113, 87%) or delayed (n=3) end-to-end anastomosis, oesophageal replacement (n=8) (6%) with gastric tube (n=4) or with jejunum interposition (n=4), and closure of the trachea-oesophageal fistula (Type E, n=9). Oesophageal continuity was achieved in all patients. Overall mortality was 3/130 (2%) and caused by gastric perforation (n=1), prolonged apnoeic spell (n =1), and food asphyxiation (n=1). Oral feeds were achieved in 121 (94%) children. Eight (6%) children remain dependent on feeding ostomy. Long-gap OA was a major predictor of post-repair complications.
Conclusion:
The modern outcome for infants with OA is characterized by an extremely low hospital mortality and satisfactory oesophageal function, enabling full oral feeds in the vast majority of children.
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