Spontaneous spinal epidural haematoma. A case report
1Department of Neurosurgery, Località Tre Conche Hospital; Avezzano, Italy - rossella.rispoli@libero.it.
The Neuroradiology Journal
|October 24, 2013
Summary
Spinal epidural hematomas are rare, often requiring emergency care. Prompt diagnosis and surgical decompression in this case led to significant recovery from sudden paraplegia.
Area of Science:
- Neurology
- Neurosurgery
- Radiology
Background:
- Spinal epidural hematomas (SEHs) are uncommon, typically resulting from trauma or medical procedures.
- Spontaneous SEH is a rare condition necessitating urgent intervention.
- Early detection and treatment are crucial for minimizing patient morbidity.
Purpose of the Study:
- To report a case of spontaneous spinal epidural hematoma presenting as sudden paraplegia.
- To highlight the importance of timely diagnosis and surgical management in improving neurological outcomes.
Main Methods:
- A 60-year-old male patient presented with acute paraplegia.
- Magnetic resonance imaging (MRI) of the thoracolumbar spine revealed an epidural mass compressing the spinal cord.
- The patient underwent emergent decompressive surgery.
Main Results:
- The MRI confirmed an epidural mass from T11 to L1 causing significant spinal cord compression.
- Post-operative surgical decompression led to substantial improvement in the patient's neurological deficits.
- The patient experienced a significant recovery from paraplegia.
Conclusions:
- Spinal epidural hematomas, though rare, can cause severe neurological deficits.
- Prompt diagnosis via MRI and emergent surgical decompression are vital for favorable outcomes in spinal epidural hematoma cases.
- This case underscores the effectiveness of rapid intervention in managing spontaneous spinal epidural hematomas.
Related Concept Videos
Hemorrhagic Stroke ll: Pathophysiology
57
A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
57
Local Anesthetics: Clinical Application as Epidural Anesthesia
1.1K
Epidural anesthetics are administered in the fat-filled epidural space, the outermost part of the spinal canal. This technique is commonly employed for pain management and anesthesia during lower abdomen and pelvis surgeries or labor and delivery.
Since epidural anesthetics can be infused through an epidural catheter, all types of drugs, including short-acting ones, can be administered. Chloroprocaine and lidocaine are examples of short and long-duration anesthetics, respectively. Bupivacaine...
Since epidural anesthetics can be infused through an epidural catheter, all types of drugs, including short-acting ones, can be administered. Chloroprocaine and lidocaine are examples of short and long-duration anesthetics, respectively. Bupivacaine...
1.1K
Hemorrhagic Stroke l: Introduction
41
A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...
41
Secondary Spinal Cord Injury llI: Pathophysiology
72
Early Ischemia and Ionic ImbalanceWithin minutes of spinal cord injury, a secondary cascade begins, progressing over hours to weeks. Vascular damage reduces blood flow, causing ischemia and mitochondrial dysfunction. ATP depletion leads to ion pump failure, membrane depolarization, sodium influx, potassium efflux, and water accumulation, resulting in cellular swelling. Increased intracellular calcium further disrupts mitochondria and accelerates cellular injury.Excitotoxicity and Neuronal...
72


