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Updated: Apr 15, 2026

Neuronavigation and Laparoscopy Guided Ventriculoperitoneal Shunt Insertion for the Treatment of Hydrocephalus
Published on: October 14, 2022
Bilateral papilledema as the first sign of ventriculoperitoneal shunt dysfunction§
Juan Carlos Serna-Ojeda1, Montserrat Aguirre-Mireles1, Mayra Fabiola Camargo-Suarez1
1Instituto de Oftalmologia #x201C;Conde de Valenciana". Mexico City, DF, Mexico.
Insights
A 12-year-old girl experienced worsening vision due to a malfunctioning ventriculoperitoneal shunt. This case highlights ophthalmologic symptoms as an early indicator of shunt dysfunction in pediatric patients.
Area of Science:
- Neuroscience
- Ophthalmology
- Pediatric Medicine
Background:
- Ventriculoperitoneal shunts are common treatments for hydrocephalus in children.
- Shunt malfunction can lead to serious neurological complications.
- Ophthalmologic findings are sometimes associated with increased intracranial pressure.
Purpose of the Study:
- To report a case of pediatric ventriculoperitoneal shunt dysfunction presenting initially with visual decline.
- To emphasize the importance of considering shunt issues in children with unexplained visual disturbances.
Main Methods:
- Case report of a 12-year-old female patient.
- Review of clinical presentation, ophthalmologic examination, and diagnostic work-up.
- Correlation of symptoms with ventriculoperitoneal shunt status.
Main Results:
- The patient presented with progressive bilateral decreased visual acuity and bilateral papilledema.
- No other systemic signs of increased intracranial pressure were observed.
- Laboratory tests ruled out infectious or inflammatory causes.
Conclusions:
- Ophthalmologic manifestations, specifically papilledema and decreased visual acuity, can be the initial presenting signs of ventriculoperitoneal shunt dysfunction.
- Early recognition of these visual changes is crucial for timely diagnosis and management of shunt complications in pediatric patients.
Abstract:
A 12 year-old girl patient with a history of ventriculoperitoneal shunt placement at the age of 6 months presented with progressive bilateral decrease in visual acuity. Ophthalmologic examination was consistent with bilateral papilledema. No other systemic manifestations of increased intracranial pressure were evident and laboratory work-up excluded other inflammatory or infectious processes. We present here an interesting case of a patient with ophthalmologic manifestations as the initial finding of ventriculoperitoneal shunt dysfunction.
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