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Updated: Feb 8, 2026

Imaging Mycobacterium tuberculosis in Mice with Reporter Enzyme Fluorescence

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[Bisalbuminemia: A case report].

B Lefrère¹, E Dedôme¹, C Garcia-Hejl¹

¹Fédération des laboratoires, hôpital d'instruction des armées Percy, 101, avenue Henri-Barbusse, 92140 Clamart, France.

La Revue De Medecine Interne

|June 20, 2018

Summary

This summary is machine-generated.

Bisalbuminemia, a rare albumin pattern modification, can be hereditary. Diagnosis involves ruling out acquired causes and confirming genetic bisalbuminemia through electrophoresis, especially when unexplained hypereosinophilia is present.

Keywords:

Bisalbuminemia Bisalbuminémie Drug interference Interférence médicamenteuse Serum protein electrophoresis Électrophorèse des protéines sériques

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Area of Science:

Biochemistry
Clinical Diagnostics

Background:

Bisalbuminemia represents rare qualitative alterations in serum albumin patterns.
Most bisalbuminemia cases are benign and detectable via capillary electrophoresis.

Observation:

A case of hereditary bisalbuminemia was incidentally discovered during the investigation of chronic hypereosinophilia in a 42-year-old patient.
Key indicators for genetic bisalbuminemia included a normal lipid profile, absence of antibiotic treatment or pancreatic issues, and persistent bisalbuminemia over a month.

Findings:

Hereditary bisalbuminemia can be diagnosed by excluding acquired etiologies like analytical interference, drug effects, pancreatitis, or immunoglobulin binding.
A systematic approach involving lipemic index check, medical history review, and repeat electrophoresis aids in identifying the cause.

Implications:

This highlights the importance of considering genetic bisalbuminemia in unexplained hypereosinophilia cases.
Accurate diagnosis of bisalbuminemia relies on differentiating genetic from acquired forms through careful clinical and laboratory evaluation.