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[Comparative evaluation of Yersinia interaction with HEp-2 cells]

Zhurnal Mikrobiologii, Epidemiologii I Immunobiologii
|December 1, 1986
PubMed

Insights

Yersinia bacteria invasion into epithelial cells is driven by bacterial cytotoxicity. This cytotoxicity, most potent in Yersinia pseudotuberculosis, influences intracellular multiplication and causes distinct cytopathic effects in host cells.

Area of Science:

  • Microbiology
  • Cell Biology
  • Pathogenesis

Background:

  • Yersinia species are significant bacterial pathogens.
  • Understanding Yersinia invasion mechanisms is crucial for disease control.

Purpose of the Study:

  • To comparatively characterize the invasion mechanisms and cytotoxic actions of Yersinia pseudotuberculosis and Yersinia enterocolitica strains.
  • To correlate bacterial cytotoxicity with intracellular multiplication and cytopathic effects.

Main Methods:

  • Optical and electron microscopy of infected HEp-2 cells.
  • Cytoenzymological analysis of cellular changes.
  • Infection assays with various Yersinia strains.

Main Results:

  • Bacterial cytotoxicity directly correlates with Yersinia invasion and intracellular multiplication.
  • Yersinia pseudotuberculosis (serovar 1) exhibited the highest cytotoxicity, followed by Yersinia enterocolitica 09, and then Yersinia enterocolitica 03.
  • Cytopathic effects included vacuolization, cytoplasm expulsion, organelle loss, autophagosome formation, nuclear changes, and cell detachment.

Conclusions:

  • Bacterial cytotoxicity is a key determinant of Yersinia's ability to invade and multiply within epithelial cells.
  • Distinct cytopathic changes and altered enzymatic activities (increased phosphatases, inhibited respiration/dehydrogenase) are associated with Yersinia infection.

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