Related Experiment Video
Updated: Jun 11, 2025
05:39
A Neonatal BALB/c Mouse Model of Necrotizing Enterocolitis
Published on: November 30, 2021
3.5K
Gastric Necrosis in a Previously Healthy Child: A Case Report
Shahnam Askarpour1, Hazhir Javaherizadeh2, Mahboobeh Rashidi3
1Department of Pediatric Surgery, Children's Medical Center, Ahvaz Jundishapur University of Medical Sciences, Ahvaz, Iran.
Middle East Journal of Digestive Diseases
|October 10, 2024
Summary
A rare pediatric surgical emergency, gastric necrosis, occurred in a 13-year-old boy. This case highlights a potential link between coronavirus infection and gastric necrosis, requiring surgical intervention and leading to complications.
Area of Science:
- Pediatric Surgery
- Infectious Diseases
- Gastroenterology
Background:
- Gastric necrosis is an extremely rare surgical emergency in children.
- Early diagnosis and surgical intervention are critical for patient outcomes.
Observation:
- A previously healthy 13-year-old boy presented with severe abdominal pain and coffee-ground vomiting.
- Physical examination revealed peritonitis and septic shock, necessitating emergency laparotomy.
- Intraoperative findings included gastric necrosis, esophageal and duodenal ischemia, and distention.
Findings:
- The patient underwent total gastrectomy and Roux-en-Y esophagojejunostomy.
- Chest CT revealed bilateral lung opacities, consistent with pneumonia.
- Reverse transcription polymerase chain reaction (RT-PCR) for coronavirus eventually tested positive.
- Severe anastomotic strictures developed during follow-up.
Implications:
- This case suggests a possible association between coronavirus infection and pediatric gastric necrosis.
- Highlights the importance of considering infectious etiologies in surgical emergencies.
- Underscores the potential for long-term complications, such as anastomotic strictures, after extensive gastrointestinal surgery.
Related Concept Videos
01:25Peptic Ulcer Disease V: Surgical Management and Nursing Care
236
Surgical management and nursing care are crucial in treating Peptic Ulcer Disease (PUD). Here is an organized and enhanced overview of the surgical interventions and the associated nursing care for PUD:
Surgical Interventions for Peptic Ulcer Disease
Surgical Interventions for Peptic Ulcer Disease
236
01:17Gastritis-II: Pathophysiology
264
Gastritis is marked by disruption of the mucosal barrier that usually protects the stomach tissue from digestive juices and manifests in acute and chronic forms.
In acute gastritis, the gastric mucosa becomes swollen and red and undergoes superficial erosion. Superficial ulceration may lead to bleeding.
In chronic gastritis, persistent or repeated insults lead to chronic inflammatory changes and, eventually, thinning or atrophy of the gastric tissue.
Gastritis can stem from various causes, each...
In acute gastritis, the gastric mucosa becomes swollen and red and undergoes superficial erosion. Superficial ulceration may lead to bleeding.
In chronic gastritis, persistent or repeated insults lead to chronic inflammatory changes and, eventually, thinning or atrophy of the gastric tissue.
Gastritis can stem from various causes, each...
264
01:23Gastritis III: Clinical Manifestations and Management
170
The clinical manifestations of gastritis can vary depending on the cause and type of gastritis, but some common symptoms may include the following.
Clinical manifestations of acute gastritis
The patient with acute gastritis may have a rapid onset of symptoms, such as epigastric pain or discomfort, dyspepsia, anorexia, hiccups, or nausea and vomiting, which can last from a few hours to a few days. Erosive or hemorrhagic gastritis may cause bleeding, which may manifest as blood in vomit or as...
Clinical manifestations of acute gastritis
The patient with acute gastritis may have a rapid onset of symptoms, such as epigastric pain or discomfort, dyspepsia, anorexia, hiccups, or nausea and vomiting, which can last from a few hours to a few days. Erosive or hemorrhagic gastritis may cause bleeding, which may manifest as blood in vomit or as...
170
01:27Gastritis-I: Introduction and Types
284
Gastritis, defined by the inflammation or irritation of the stomach lining or gastric mucosa, manifests in several distinct forms: acute, chronic, reactive, and a specific subtype known as autoimmune metaplastic atrophic gastritis.
Acute gastritis presents as a sudden inflammation triggered by various stressors to the stomach lining, such as exposure to corrosive agents, local irritants like aspirin and other NSAIDs, alcohol consumption, radiation therapy, physical trauma, severe burns, sepsis,...
Acute gastritis presents as a sudden inflammation triggered by various stressors to the stomach lining, such as exposure to corrosive agents, local irritants like aspirin and other NSAIDs, alcohol consumption, radiation therapy, physical trauma, severe burns, sepsis,...
284
01:30Esophageal Strictures-I: Introduction
66
Esophageal strictures involve abnormal narrowing or tightening of the esophagus. They vary in length and severity, ranging from mild constriction to complete obstruction, and are classified as benign (noncancerous) or malignant (cancerous).
Etiology
The primary cause of esophageal strictures is long-standing gastroesophageal reflux disease (GERD), accounting for about 70 to 80% of adult cases. Chronic acid reflux can lead to injury and scarring of the esophageal lining, culminating in...
Etiology
The primary cause of esophageal strictures is long-standing gastroesophageal reflux disease (GERD), accounting for about 70 to 80% of adult cases. Chronic acid reflux can lead to injury and scarring of the esophageal lining, culminating in...
66
01:28Esophageal Perforation-II: Clinical Manifestations and Management
44
Esophageal perforations manifest in various clinical forms, influenced by factors such as the perforation's cause and location (cervical, intrathoracic, or intra-abdominal), the extent of contamination, and potential injury to adjacent mediastinal structures. The timing between the perforation occurrence and treatment initiation also affects the clinical presentation.
Clinical Manifestations:
Clinical Manifestations:
44