Related Experiment Video
Updated: Feb 12, 2026
Robot Assisted Distal Pancreatectomy with Celiac Axis Resection DP-CAR for Pancreatic Cancer: Surgical Planning and Technique
Published on: August 14, 2021
Evaluation of Pancreatic Steatosis in Patients with Celiac Disease
Ahmet Tarhan1, Yasemin Gökden2, Sinem Aydemir3
1Department of Internal Medicine, Health Sciences University Prof.Dr. Cemil Taşcıoğlu City Hospital, İstanbul, Türkiye.
Background/Aims:
Celiac disease, a chronic autoimmune disorder, has been reported to be associated with pancreatic involvement, including exocrine pancreatic insufficiency, pancreatitis, and cystic fibrosis. The aim of this cross-sectional study was to determine the frequency of pancreatic steatosis (PS) in patients with celiac disease and compare it with that in healthy controls.
Materials And Methods:
Sixty patients with celiac disease and 60 healthy participants were included in this study. Biochemical and hematological parameters were collected from all participants. Hepatic steatosis (HS) and PS were diagnosed by ultrasonography and were compared between the groups.
Results:
Age, gender, and body mass index were similar between the groups (P > .05). Pancreatic steatosis (81.7%) and HS (66.7%) were more prevalent in the celiac group compared to healthy controls (P < .001). A positive and significant correlation was found between PS and HS in the celiac group (rho = 0.464, P < .05). Hepatic steatosis and PS did not differ between tissue transglutaminase Immunoglobulin A (IgA)-positive and -negative patients with celiac disease (P > .05). No differences in HS or PS were found between celiac patients who adhered to a gluten-free diet and those who did not (P > .05).
Conclusion:
Celiac disease may be associated with an increased risk of HS and PS. Cite this article as: Tarhan A, Gökden Y, Aydemir S, Önol S, Özek FZ. Evaluation of pancreatic steatosis in patients with celiac disease. Turk J Gastroenterol. 2026;37(2):233-241.
Related Concept Videos
01:21Peripheral Arterial Disease II: Clinical Manifestations and Diagnostic Evaluation
01:20Nursing Process for Patient and Caregiver Teaching III: Evaluation and Documentation
Nurses can use several methods to evaluate patient outcomes. For example, oral questions can assess cognitive learning,...
01:26Pancreatic Juice and Secretion
When acidic chyme from the stomach enters the duodenum, it triggers the release of secretin, a hormone that prompts pancreatic juice secretion. After a fatty meal, cholecystokinin, another hormone, stimulates gallbladder contraction and enhances enzyme-rich...
01:27Acute Pancreatitis I: Introduction
Acute pancreatitis is characterized by rapid inflammation of the pancreas, often caused by factors like gallstone blockage or excessive alcohol consumption. Chronic pancreatitis, on the other hand, is a slow, progressive inflammation that may result from long-term alcohol abuse, obstructions in the pancreatic duct, or genetic factors.
The causes of acute pancreatitis include:
01:24Chronic Pancreatitis I: Introduction
Pancreatitis is the inflammation of the pancreas, which occurs when the immune system becomes active and causes swelling, pain, and disruptions in organ function. Pancreatitis can manifest as either an acute or chronic condition.
Acute pancreatitis arises suddenly and lasts for a brief duration, while chronic pancreatitis is a long-term affliction...
01:29Chronic Pancreatitis II: Collaborative Care
Assessment: