Related Experiment Videos
Independent Relationship between Incarceration Factors and Glycemic Control in African Americans with Criminal Legal
Objective:
Examine the relationship between incarceration factors and glycemic control in African Americans with criminal legal involvement and type 2 diabetes mellitus (T2DM).
Methods:
Using a cross-sectional survey analysis of 171 adults, a model was estimated using stepwise regression with forward selection to evaluate the relationship between incarceration factors and glycemic control, controlling for demographic and clinical factors.
Results:
Reporting a drug-related conviction (β:-0.81; 95%CI:-1.50,-0.11) was associated with higher HbA1c compared with no drug-related conviction. Compared with using only pills or oral hypoglycemic agents, the combination of pills/oral hypoglycemic agents and insulin (β:1.31; 95%CI:0.60,2.01) and using insulin or injectables alone (β:0.94; 95%CI:0.14,1.74) were associated with higher HbA1c. Each additional comorbidity (β:-0.18; 95%CI:-0.33,-0.04) and unit increase in age (β:-0.04; 95%CI:-0.08,-0.01) was associated with a lower HbA1c.
Conclusion:
History of a drug-related conviction, age, comorbidity, and medication were associated with glycemic control in African Americans with a history of criminal legal involvement and T2DM.
Related Concept Videos
Diabetes Mellitus: Type 2 and Gestational
Diabetes: Symptoms, Diagnosis, and Complications
Diabetes Mellitus: Overview and Type I Subtype
Type 1 diabetes is an autoimmune disease in which the immune system mistakenly attacks and destroys the insulin-producing beta cells in the pancreas. As a result, the body is unable to produce sufficient insulin, and individuals with...
Type II Diabetes I: Introduction
Carbohydrate Metabolism
Starch accounts for approximately 60% of the carbohydrates consumed by humans. Since amylase enzymes cannot function in the stomach's acidic environment, starch can only be digested in the mouth and small intestine. Simple sugars are found naturally in milk and fruits in the...
Type II Diabetes II: Pathophysiology