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[Circulatory disturbances in the embryo (author's transl)]
Circulatory disturbances in human embryo in the phase of organogenesis are frequent, as shown by the pathological-anatomical findings and the results of experimental embryology. There is edema and hemorrhage around the blood vessels and emboli of necrotic tissue (chiefly from the chorion) are found. The embryonic heart, which shows peristaltic movements, has a very small reserve of performance. Since there is no peripheral regulation of circulation, every damage to the vascular system results in a higher cardiac load. During organogenesis the extraembryonic vascular plexus is considerably larger than the intraembryonic plexus. Lesions of the chorion, the barrier between embryo and environment, can result in chronic or acute cardiac insufficiency. This is presumably one of the reasons why the mortality in this developmental stage is particularly high.
Circulatory disturbances in human embryo in the phase of organogenesis are frequent, as shown by the pathological-anatomical findings and the results of experimental embryology. There is edema and hemorrhage around the blood vessels and emboli of necrotic tissue (chiefly from the chorion) are found. The embryonic heart, which shows peristaltic movements, has a very small reserve of performance. Since there is no peripheral regulation of circulation, every damage to the vascular system results in a higher cardiac load. During organogenesis the extraembryonic vascular plexus is considerably larger than the intraembryonic plexus. Lesions of the chorion, the barrier between embryo and environment, can result in chronic or acute cardiac insufficiency. This is presumably one of the reasons why the mortality in this developmental stage is particularly high.