Related Experiment Video

Updated: Aug 3, 2026

Reproducible Motor Deficit Following Aortic Occlusion in a Rat Model Of Spinal Cord Ischemia
08:10

Reproducible Motor Deficit Following Aortic Occlusion in a Rat Model Of Spinal Cord Ischemia

Published on: July 22, 2017

Reversible motor and sensory neurophysiological abnormalities in cauda equina claudication

I K Saadeh1, L S Illis, A R Jamshidi Fard

  • 1Wessex Neurological Centre, Southampton University Hospital, UK.

Journal of Neurology, Neurosurgery, and Psychiatry
|October 1, 1994
PubMed
Summary

This study presents a case of cauda equina claudication, showing reversible neurophysiological changes. Central motor conduction time (CMCT) was prolonged during symptoms, offering new insights into nerve ischemia.

More Related Videos

Spinal Cord Lateral Hemisection and Asymmetric Behavioral Assessments in Adult Rats08:46

Spinal Cord Lateral Hemisection and Asymmetric Behavioral Assessments in Adult Rats

Published on: March 24, 2020

Assessing Functional Recovery of Eupneic Diaphragm Activity Following Unilateral Cervical Spinal Cord Hemisection in Rats05:09

Assessing Functional Recovery of Eupneic Diaphragm Activity Following Unilateral Cervical Spinal Cord Hemisection in Rats

Published on: June 14, 2024

Related Concept Videos

Secondary Spinal Cord Injury llI: Pathophysiology01:25

Secondary Spinal Cord Injury llI: Pathophysiology

Early Ischemia and Ionic ImbalanceWithin minutes of spinal cord injury, a secondary cascade begins, progressing over hours to weeks. Vascular damage reduces blood flow, causing ischemia and mitochondrial dysfunction. ATP depletion leads to ion pump failure, membrane depolarization, sodium influx, potassium efflux, and water accumulation, resulting in cellular swelling. Increased intracellular calcium further disrupts mitochondria and accelerates cellular injury.Excitotoxicity and Neuronal...