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Cellular and Molecular Life Sciences : CMLS|May 3, 2014
Processing of heparanase is mediated by syndecan-1 cytoplasmic domain and involves syntenin and α-actininAnna Shteingauz, Neta Ilan, Israel VlodavskyAutophagy|November 17, 2015
Function from within: Autophagy induction by HPSE/heparanase--new possibilities for interventionNeta Ilan, Anna Shteingauz, Israel VlodavskyJournal of Cell Science|February 19, 2009
The BTB-MATH protein BATH-42 interacts with RIC-3 to regulate maturation of nicotinic acetylcholine receptorsAnna Shteingauz, Emiliano Cohen, Yoav Biala, et al.Cancer Research|August 8, 2015
Heparanase Enhances Tumor Growth and Chemoresistance by Promoting AutophagyAnna Shteingauz, Ilanit Boyango, Inna Naroditsky, et al.Journal of Leukocyte Biology|December 10, 2013
Modification of heparanase gene expression in response to conditioning and LPS treatment: strong correlation to rs4693608 SNPOlga Ostrovsky, Avichai Shimoni, Polina Baryakh, et al.The Journal of Biological Chemistry|June 26, 2010
Heparanase 2 interacts with heparan sulfate with high affinity and inhibits heparanase activityFlonia Levy-Adam, Sari Feld, Victoria Cohen-Kaplan, et al.Seminars in Thrombosis and Hemostasis|February 16, 2015
Latent heparanase facilitates VLA-4-mediated melanoma cell binding and emerges as a relevant target of heparin in the interference with metastatic progressionUrsula Gerber, Sebastian G Hoß, Anna Shteingauz, et al.Cell Death & Disease|October 21, 2018
AMPK-dependent autophagy upregulation serves as a survival mechanism in response to Tumor Treating Fields (TTFields)Anna Shteingauz, Yaara Porat, Tali Voloshin, et al.Journal of Visualized Experiments : Jove|May 19, 2017
Determining the Optimal Inhibitory Frequency for Cancerous Cells Using Tumor Treating Fields (TTFields)Yaara Porat, Moshe Giladi, Rosa S Schneiderman, et al.Frontiers in Oncology|July 12, 2021
<i>In Vivo</i> Safety of Tumor Treating Fields (TTFields) Applied to the TorsoRoni Blatt, Shiri Davidi, Mijal Munster, et al.Pageof 2