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Current Opinion in Genetics & Development|February 1, 1994
Myc-Max-Mad: a transcription factor network controlling cell cycle progression, differentiation and deathB Amati, H LandBiochimica Et Biophysica Acta|August 8, 1996
AACR-ISREC joint symposium "Cancer and the cell cycle' January 17-20, 1996, Lausanne, SwitzerlandB Amati, M PeterMolecular and Cellular Biology|October 1, 1990
Drosophila scaffold-attached regions bind nuclear scaffolds and can function as ARS elements in both budding and fission yeastsB Amati, S M GasserThe EMBO Journal|October 6, 1997
Phosphorylation-dependent degradation of the cyclin-dependent kinase inhibitor p27J Vlach, S Hennecke, B AmatiCell|September 23, 1988
Chromosomal ARS and CEN elements bind specifically to the yeast nuclear scaffoldB B Amati, S M GasserOncogene|May 10, 2000
Conserved region 2 of adenovirus E1A has a function distinct from pRb binding required to prevent cell cycle arrest by p16INK4a or p27Kip1K Alevizopoulos, B Sanchez, B AmatiFrontiers in Bioscience : a Journal and Virtual Library|February 27, 1998
Myc and the cell cycleB Amati, K Alevizopoulos, J VlachNucleic Acids Research|December 22, 1999
Myc induces the nucleolin and BN51 genes: possible implications in ribosome biogenesisP J Greasley, C Bonnard, B AmatiNucleic Acids Research|November 25, 1993
Distinct DNA binding preferences for the c-Myc/Max and Max/Max dimersD L Solomon, B Amati, H LandThe EMBO Journal|October 6, 1997
Cyclin E and c-Myc promote cell proliferation in the presence of p16INK4a and of hypophosphorylated retinoblastoma family proteinsK Alevizopoulos, J Vlach, S Hennecke, et al.Pageof 4